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The decomposition of sodium chlorite was studied in a well‐stirred reactor in a pH range of 2—3 and a temperature range of 15—35°C varying the chlorite concentration. The reaction rate was evaluated by analyzing the consumption of chlorite iodometrically. The rate law was established. Reaction was found to be of the first order with respect to both chlorite and acid concentration. Temperature dependence of the reaction was also studied and pre‐exponential Arrehenius parameter and activation energy were determined. The optimum conditions to generate chlorine dioxide from sodium chlorite are also highlighted.

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The cornea is a transparent and avascular tissue that plays a central role in light refraction and provides a physical barrier to the external environment. Corneal avascularity is a unique histological feature that distinguishes it from the other parts of the body. Functionally, corneal immune privilege critically relies on corneal avascularity. Corneal lymphangiogenesis is now recognized as a general pathological feature in many pathologies, including dry eye disease (DED), corneal allograft rejection, ocular allergy, bacterial and viral keratitis, and transient corneal edema. Currently, sizable data from clinical and basic research have accumulated on the pathogenesis and functional role of ocular lymphangiogenesis. However, because of the invisibility of lymphatic vessels, ocular lymphangiogenesis has not been studied as much as hemangiogenesis. We reviewed the basic mechanisms of lymphangiogenesis and summarized recent advances in the pathogenesis of ocular lymphangiogenesis, focusing on corneal allograft rejection and DED. In addition, we discuss future directions for lymphangiogenesis research.