Effective nutrition educations for prevention of chronic diseases for the general population are of great importance these days. The purpose of this study was to evaluate the feasibility of nutrition education for cardiovascular risk factor reduction by e-mail education in male workers. The participants were divided into three groups by age; 28-39 age group, 40-49 age group, and 50-59 age group who got regular checkups for anthropometry and biochemistry. The 1 year program consisted of 15 topics containing information about metabolic syndrome (MS) and healthy eating behavior (intake of salt, fat and alcohol). Seven hundred thirty nine participants volunteered for the study [28-39 age group, n = 240; body mass index (BMI) = 24.9 ± 2.7 kg/m 2 : 40' group, n = 276; BMI = 24.8 ± 2.6 kg/m 2 : 50' group, n = 223; BMI = 24.9 ± 2.7 kg/m 2 ]. Percentage body fat (p < 0.05) and percentage of abdominal fat (p < 0.05), total cholesterol (p < 0.05), systolic blood pressure (p < 0.05), and diastolic blood pressure (p < 0.05) were significantly decreased in all participants after the 1 year program. The total number of participants who had MS was decreased from 216 to 199 and especially the incidence of MS was decreased 27% in the group of subjects who were under the age 39. The e-mail worksite nutrition education program shows a substantial contribution to the development of effective CVD and chronic disease control and lifestyle nutrition educations that are applicable to and attractive for the large population at risk.
The purpose of this research was to investigate the influence of alcohol consumption and smoking habits on atherogenic index, antioxidant enzymes, level of lipid peroxidation and lymphocyte damage. The participants included 386 men 19–60 years of age. The level of lipid peroxidation was measured as the level of conjugated diene (CD) and the lymphocyte DNA damage was measured by COMET assay. In the observed sample population, we categorized the subjects into 15 groups. Subjects were divided into 5 groups: A0 (0 g/day, n=49), A1 (1–70 g/day, n=98), A2 (71–140 g/day, n=84), A3 (141–350 g/day, n=101), A4 (>350 g/day, n=40), and then were further divided into S0 (nonsmoker, n=92), S1 (1–15 cigarettes/day, n=138), S2 (>15 cigarettes/day, n=142) depending on the level of cigarette consumption. Regardless of the alcohol intake, there was a continuous increase of the atherogenic index with the increasing quantity of cigarette smoking. HDL‐C was increased with the increasing level of alcohol consumption. In addition, regardless of the alcohol intake, CD level was lowest in the S0 groups and highest in the S3 groups. Antioxidant status measured by plasma tocopherol, carotene and vitamin C level did not differ significantly among groups. The DNA damage (assessed by tail DNA) was greatest in the heavy smokers compared to the non‐ or light smokers. Our result showed that the exposure of cigarette smoking has higher impact on the level of antioxidants and DNA damage than the combined effect with alcohol consumption.This study was supported by a grant from Kyung Hee University Research Fund in 2004 (KHU‐20040945)
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