Although there are some Swedish studies giving reference values for height and weight in adulthood, no values of weight for height (W/H) are available. W/H is an essential measurement, as it permits comparison of weight independent of the height of an individual. We here present a method of constructing W/H standards, illustrated by a material sample of 4438 healthy individuals, 20-80 years of age, living in the Uppsala County during the period 1964-1971. In setting standards of W/H, the 'normal' increase in weight during adulthood and the distribution skewness of weight are both shown to be important. This weight gain starts at about 40 years of age, and is completed at approximately 55 years of age. It can be regarded as a 'normal' pattern, but not necessarily the optimum healthy one. Using ordinary growth charts for weight and height, a simple means of expressing W/H in standard deviation scores is outlined. It is difficult to find a representative Swedish sample, owing to geographical and psychosocial differences, and also to the secular trend phenomenon. However, the mean values for weight and height obtained in the present study fall between the mean values of two other recently conducted Swedish studies.
Summary Acute cerebellar ataxia is a syndrome with heterogeneous etiology. The present paper describes 6 cases. Three cases exhibited cerebellar‐ataxic symptoms only, two had symptoms indicating that the process affected the cerebrum also, and in one case there were also spinal symptoms (paresis). In three cases the causation was unquestionably polio infection with significant serological tests and pareses. In two of the remaining cases the infectious agent possibly was influenza B, and in the third case, possibly influenza A was responsible. The difference in the severity of the clinical picture between the unquestionable polio cases and the other cases is pointed out. The difficulty of differentiating between tumor of the posterior fossa or the brain stem is illustrated by an additional case, in which the onset initially was in complete Agreement with the symptomatology of acute cerebellar ataxia. Ataxie cérébelleuse aiguë chez l'enfant. L'auteur rapporte six cas, dont trois présentaient uniquement des symptômes d'ataxie cérébelleuse, deux avaient de plus des symptômes de lésions cérébrales et un avait même des signes d'atteinte médullaire (parésie). Dans trois cas la cause était une infection manifeste de poliomyélite (mise en évidence du virus, parésie). Dans deux autres cas l'agent pathogène était peut‐êret l'Influenza B et dans un troisième cas peut‐être l'Influenza A. L'auteur insiste sur les différences qui existent dans le tableau clinique et son degré de gravité. Pour illustrer la difficulté de faire le diagnostic différentiel avec une tumeur intracranienne de la fosse postérieure et du bulbe, il rapporte un cas où les symptômes du début correspondaient à ceux d'une ataxie cérébelleuse. Akute Kleinhirnataxie bei Kindern. Besprechung von 6 Fällen, von denen drei nur Symptome von Kleinhirnataxie, zwei gleichzeitig andere Symptome (cerebrale Läsionen) zeigten und ein Fall, bei dem sogar spinale Zeichen (Paresen) vorhanden waren. In den 3 Fallen war die Ursache offensichtlich eine Poliomyelitisinfektion (Virusnachweis, Paresen). In den anderen Fallen war die Infektion wahrscheinlich ausgelöst durch Influenza B und im 3. Fall wahrscheinlich durch Influenza A. Die Unterschiede im klinischen Bild und der Schwerheitsgrad werden betont. Die Schwierigkeiten der Differentialdiagnose gegen über einem intrakranialen Tumor im Beroich der Fossa posterior oder des Hirn‐stammes werden durch Besprechung eines Falles dargestellt, bei dem die Krankheits‐zeichen und Symptome anfänglich mit denen einer cerebellaren Ataxie überein‐stimmten. Ataxia cerebelosa aguda en niños. Se refieren seis casos: tres de ellos tenían solamente síntomas de ataxia cerebelosa, dos de ellos síntomas adicionales y uno de ellos lesiones cerebrales y signos medulares (paresia). En tres casos fué una evidente infección a polio (se demostró el virus, paresias). En los otros casos la infección fué producida posiblemente por virus B de Influenza, en el tercer caso posiblemente por el virus Influenza A. Se pusieron en evidencia diferencias en el cuadro ...
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