Objective
To assess the prostheses and implants survival rate and peri‐implantitis rate in edentulous patients treated with full‐arch screw‐retained implant‐supported fixed dental prostheses (FSIFDPs) and full‐arch telescopic‐retained implant‐supported fixed dental prostheses (FTIFDPs) over an observation period of at least 5 years.
Materials and methods
From 2004 to 2012, 696 implants were inserted into 78 patients with 102 prostheses. The FSIFDP group comprised 31 patients (37 prostheses, 232 implants), whereas the FTIFDP group comprised 47 patients (65 prostheses, 464 implants). Prosthesis and implant estimated cumulative survival rates (ECSR) and estimated cumulative peri‐implantitis rates (ECPR) were assessed. The follow‐up period was 5–12 years. Kaplan–Meier survival curves with the log‐rank test were used to evaluate outcomes.
Results
The 12‐year prosthesis ECSR was 96.8% (95% CI: 79.2–99.5, 36/37 prostheses) in the FSIFDP group and 96.4% (95% CI: 86.3–99.1, 63/65 prostheses) in the FTIFDP group, whereas the 12‐year implant ECSR was 99.5% (95% CI: 96.4–99.9, 231/232 implants) in the FSIFDP group and 98.7% (95% CI: 96.9–99.5, 459/464 implants) in the FTIFDP group. The 12‐year ECPR at the prosthesis level was 12.8% (95% CI: 12.7–47.6, 4/37 prostheses) in the FSIFDP group and 12.8% (95% CI: 11.4–24.1, 6/65 prostheses) in the FTIFDP group. The 12‐year ECPR at the implant level was 4.4% (95% CI: 4.3–23.0, 6/232 implants) in the FSIFDP group and 2.2% (95% CI: 2.0–12.3, 7/464 implants) in the FTIFDP group.
Conclusion
FTIFDPs have clinical results comparable to those of FSIFDPs. Therefore, FTIFDPs can be useful.
Objective
The mechanisms underlying the onset and progression of peri-implantitis are similar to those of periodontitis, and the causative bacteria are believed to similar. Previous studies support an association between peri-implantitis and periodontal pathogen. Thus, we investigated the bacterial flora of peri-implantitis patients in comparison to those of healthy implant and periodontitis patients.
Materials and methods
In total, 70 patients visiting Tokyo Dental College Chiba Hospital were divided into four groups: healthy, periodontitis, healthy implant, and peri-implantitis. For each group, the following five periodontal pathogens were detected using real-time polymerase chain reaction: Porphyromonas gingivalis, Aggregatibacter actinomycetemcomitans, Tannerella forsythia, Treponema denticola, and Prevotella intermedia.
Results
The average copy number of total bacteria was significantly higher in the periodontitis group than in the other groups. P. gingivalis was detected in the periodontitis and peri-implantitis groups at levels as high as 18.92% and 12.29%, respectively, and P. intermedia was found in the peri-implantitis group at a rate of 2.06%. Nevertheless, periodontal pathogens were generally detected at lower levels in the peri-implantitis group than in the periodontitis group.
Conclusion
We found lower bacterial counts in the peri-implantitis group relative to the periodontitis group. Our results suggest that the peri-implant tissue is less resistant to bacteria, so even a small number of bacteria can be a risk factor for peri-implantitis and the causative agent of peri-implantitis can be bacteria other than periodontal pathogen.
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