Facial palsy in children is a manifestation of a heterogeneous group of causes. The most common aetiology of FNP in children in our study was idiopathic (Bell's palsy), followed by infective causes, such as acute otitis media and neurotropic herpes viruses. Therefore, treatment should be adapted to each patient depending on the underlying disease and severity of FNP.
Patient: Male, 62Final Diagnosis: Gallbladder hypoplasiaSymptoms: Abdominal painMedication: —Clinical Procedure: —Specialty: SurgeryObjective:Congenital defects/diseasesBackground:There are different variations in the anatomy of the gallbladder. Congenital abnormalities of the gallbladder such as agenesis and hypoplasia are rare conditions and difficult to diagnose with imaging studies. Patients are usually asymptomatic or have symptoms that mimic gallstone disease. The diagnosis is often made intraoperatively and is established by histopathological examination.Case Report:We report a case of a 62-year-old male who had cholelithiasis symptoms and was falsely diagnosed with gallstone disease by abdominal ultrasound scan. The patient underwent an operation which revealed a rudimentary gallbladder. The histology result showed hypoplastic gallbladder tissue.Conclusions:This case suggests that surgeons need to take into consideration congenital anomalies of the gallbladder intraoperatively in order to avoid any iatrogenic injury to biliary tract during a routine laparoscopic cholecystectomy. Intraoperative cholangiography can be a useful tool to avoid unnecessary surgical risky interventions.
Background: Nasal alar deformity (NAD) can affect both nasal breathing and the aesthetic appearance of the nose. NAD can be congenital or acquired. The acquired causes include nerve palsies, traumatic or iatrogenic. A rare condition that is associated with NAD is trigeminal trophic syndrome (TTS). TTS is an uncommon disorder of the trigeminal nerve characterized by a triad of facial ulcers, with anesthesia and paresthesia of the involved dermatomes. Nasal alar reconstruction is challenging for the surgeon and when it is performed, a delicate balance between aesthetic and functional outcome should be considered. Case report: A 37 years old woman presented with nasal alar deformity caused by facial and trigeminal nerve paralysis after brain surgery. The woman was diagnosed with an acoustic neuroma and underwent complete tumor removal from neurosurgeons. After surgery, the patient suffered from facial and trigeminal nerve paralysis and 4-years later she presented with a progressive nasal alar deformity accompanied by breathing and aesthetic dysfunction. Reconstruction of the nasal ala was performed in one-stage surgery using spreader, alar batten and rim grafts. Seven years postoperatively, our patient had good nasal breathing and did not show any alar collapse. Conclusions: In our case we believe that facial and trigeminal nerve palsies as a result of brain surgery, have both contributed to the deformity of the nasal ala. Reconstruction with the optimal technique and graft guaranteed long term results.
Tenosynovial giant cell tumor (TSGCT) is a benign tumor that commonly presents in the upper limb joints and hands and less frequently occurs in the lower extremities. Usually they arise from synovium or tendon sheath. The aim of this article is to present an unusual location of TSGCT of the supraclavicular region in a young patient.
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