This article describes the results of a study of 128 cats with urological syndrome. The study was conducted to determine the incidence of this syndrome, its nosological structure, analysis of clinical signs, as well as laboratory data characteristic of this syndrome. The data were obtained from the patients of the clinical branches of the DSTU, Rostov-on-Don, the patients had not been treated anywhere before and were first seen with urological syndrome of cats. A medical history was compiled for all cats, a clinical examination was carried out, and blood and urine were collected for analysis. As a result of the studies, it was revealed that as part of the urological syndrome, idiopathic cystitis of cats occurred in 65% of cases, urinary tract infections in 14% of cats, obstruction of the urethra caused by urethral plugs was observed in 68%, urolithiasis in 28%, and only in In 0.7% of cats, proliferative changes in the urethral tract were noted. In 79% of cats with urological syndrome, it was accompanied by urethral obstruction with different mechanisms of occurrence.
Feline lower urinarytract disease (FLUTD) is a life-threatening condition in cats, especially in obstructive males. In a clinical situation, early diagnosis and correction of this condition is very important, otherwise the disease ends in death. FLUTD manifested by stranguria, pollakiuria, dysuria, and, in severe cases, hematuria and obstruction by anuria. This article discusses the features of the clinical course of FLUTD in 5 cats with obstructive idiopathic cystitis. Animals were selected with a similar history, they were animals from 3 to 7 years old, with exclusively home keeping, no walking and a diet consisting of dry commercial food. All animals underwent a complete clinical examination, complete blood count, general urinalysis, ultrasound of the genitourinary system and heart. For two weeks, the animals underwent a complex treatment consisting of infusion therapy, anesthesia with dexmedetomidine and NSAIDs (meloxicam), which led to clinical recovery in all animals within 14 days, all animals on an ongoing basis were prescribed amitriptyline, observation of the animals for 6 months did not reveal a relapse of the disease.
The left ventricular non-compaction is a disease characterizing by hyper-trabecularization of myocardium causing development of chronic heart failure. In the development of structural damages under this pathology the main role belongs to genetic disorders and dysfunction of organogenesis in early periods of gestation. The clinical manifestations are associated with development of systolic dysfunction of left ventricle, intricate abnormalities of rhythm and thromboembolic occurrences. In patients with systolic dysfunction of left ventricle phenomenon of hyper-trabecularization of myocardium is quite often found during transthoracic echocardiography. The modern diagnostic criteria permit approaching differentially issues of verification of non-compact myocardium and redundant trabecularization in patients with chronic heart failure with reduced fraction of output of left ventricle.
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