The neuropsychological assets and deficits of several types of pediatric neurological disease, disorder, and dysfunction are described. These are examined from the perspective of the syndrome of nonverbal learning disabilities (NLD) and the "white matter model" designed to explain its complex manifestations. It is concluded that children with some of these diseases exhibit the NLD phenotype, whereas others do not. For the most part, the diseases in which the NLD phenotype is particularly evident are those wherein it has been demonstrated that perturbations of white matter (long myelinated fibers) are particularly prominent.

Tables permitting the conversion of short-form composite scores to full-scale IQ estimates have been published for previous editions of the Wechsler Adult Intelligence Scale (WAIS). Equivalent tables are now needed for selected subtests of the WAIS-III. This article used Tellegen and Briggs's formulae to convert the sum of scaled scores for four selected WAIS-III short-form combinations into full-scale IQ estimates. Conversion tables providing full-scale IQ estimates across all age groups from the sum of scaled scores for these four short forms are presented. Reliability and validity estimates for these short forms are also provided for all age groups.

Objective: The Montreal Cognitive Assessment (MoCA) is a general cognitive screening tool that has shown sensitivity in detecting mild levels of cognitive impairment in various clinical populations. Although mood dysfunction is common in referrals to memory clinics, the influence of mood on the MoCA has to date been largely unexplored. Method: In this study, we examined the impact of mood dysfunction on the MoCA using a memory clinic sample of individuals with depressive symptoms who did not meet criteria for a neurodegenerative disease. Results: Half of the group with depressive symptoms scored below the MoCA-suggested cutoff for cognitive impairment. As a group, they scored below healthy controls, but above individuals with Alzheimer's disease and frontotemporal dementia. A MoCA subtask analysis revealed a pattern of executive/attentional dysfunction in those with depressive symptoms. Conclusions: This observed negative impact of depressive symptomatology on the MoCA has interpretative implications for its utility as a cognitive screening tool in a memory clinic setting. Bien qu'une dysfonction de l'humeur soit fréquente chez les patients référés à des cliniques de la mémoire, l'influence de l'humeur sur le MoCA n'a encore jamais été explorée. Méthode: Nous avons examiné l'impact d'un trouble de l'humeur sur le MoCA chez un échantillon de patients d'une clinique de la mémoire présentant des symptômes de dépression qui ne rencontraient pas les critères diagnostiques d'une maladie neurodégénérative. Résultats: La moitié du groupe présentant des symptômes de dépression avait un score sous le niveau suggéré par le MoCA pour le diagnostic d'une atteinte cognitive. En tant que groupe, leur score était inférieur à celui des témoins en bonne santé, mais supérieur à celui des patients atteints de la maladie d'Alzheimer ou de démence fronto-temporale. L'analyse des sous-tâches a montré un profil de dysfonction exécutive/de l'attention chez ceux qui présentaient des symptômes dépressifs. Conclusions: L'impact négatif de la symptomatologie dépressive sur le MoCa que nous avons observé a des implications sur l'interprétation de ce test concernant son utilité comme outil de dépistage cognitif dans le contexte d'une clinique de la mémoire.

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