Background
Clinical registry participation is a measure of healthcare quality. Limited knowledge exists on Australian hospitals participation in clinical registries and whether this registry data informs quality improvement initiatives. Hence, our study aimed to; identify participation in clinical registries; determine if registry data inform quality improvement initiatives; identify registry participation enablers; and clinicians’ educational needs to improve use of registry data to drive practice change.
Methods
: A self-administered survey was distributed to staff coordinating registries in seven hospitals in New South Wales, Australia. Eligible registries were international, national and state-based clinical, condition/disease-specific and device/product registries.
Results
: Response rate was 70% (97/139). Sixty-two (64%) respondents contributed data to 46 eligible registries. Registry reports were most often received by nurses (61%) and infrequently by hospital executives (8.4%). Less than half used registry data ‘always’ or ‘often’ to influence practice improvement (48%) and care pathways (49%). Protected time for data collection (87%) and benchmarking (79%) were ‘very likely’ or ‘likely’ to promote continued participation. Over half ‘strongly agreed’ or ‘agreed’ that clinical practice improvement training (79%) and evidence-practice gap identification (77%) would optimise use of registry data.
Conclusions
: Registry data are generally only visible to local speciality units and not routinely used to inform quality improvement. Centralised on-going registry funding, accessible and transparent integrated information systems, combined with data informed improvement science education could be first steps to promote quality data-driven clinical improvement initiatives.
Summary1. A method of recording the peristaltic frequency and the rate of transport of fluid (perfusion rate) in the rat ureter in viva is described. 2. Acetylcholine and atropine did not alter ureteral activity. Histamine increased the rate of peristalsis by up to 15% and the rate of perfusion by up to 10%. Low doses of 5-hydroxytryptamine increased peristaltic frequency whereas high doses decreased peristaltic frequency; all doses reduced the rate of perfusion. 3. Morphine reduced the rate of perfusion by 5-10% at all dose levels, but only the highest dose used reduced the frequency of ureteral peristalsis. 4. (-)-Adrenaline, (-)-noradrenaline and (±)-isoprenaline reduced the frequency of peristalsis. The order of potency was isoprenaline>noradrenaline> adrenaline. The response was dose-related and blocked by propranolol, which itself did not affect ureteral activity.
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