Summary
Two patients in whom pleurodesis was used for intractable pleural effusion due to congestive heart failure are described. This procedure eliminated the pleural effusion but resulted in accumulation of fluid in other body compartments. Pleurodesis has a limited role in the treatment of intractable pleural transudates in congestive heart failure.
Precooling to 28°C alters the course of hemorrhagic shock in the dog by prolonging the period of tolerance to severe hypotension, and by prolonging the survival time after transfusion from an average of several hours to an average of 30 hours. Precooling does not prevent death. But if an antibiotic is given at the time of transfusion all precooled dogs recover, whereas normothermic dogs given an antibiotic at the time of transfusion do not recover. Precooling protects the dog in hemorrhagic shock by sustaining the antibacterial defense mechanisms, which disintegrate rapidly in the normothermic dog. The protective effects of cooling is not secured if it is applied after the induction of hemorrhagic shock. The effects of hypothermia upon the cardiovascular dynamics are described and their significance discussed.
Transfusion for experimental hemorrhagic to transfusion is a result of bacterial activity, shock of 2 hours duration is followed by re-which cannot be controlled because shock covery. But if the shock is allowed to persist produces a progressively Severe injury to the for much longer (4-5 hours) it fails to re-antibacterial mechanisms( 1-6). The further spond to transfusion. Evidence has been given observation that intravenous antibiotic therthat the failure of the circulation to respond apy, give11 shortly after shock was induced, bility, in contrast to the Success of !XIch there 4 Supported by contract with oftice of Surgeon failed to prevent the development of irreversiapy when given before shock was induced General, U. S. Army, and by research grant from SIH.
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