Ever since probiotics have become a modern topic to discuss in the last few decades, they came out as the most important and independent regulatory system of our metabolism. ``We are what we eat`` or the way of how food modeling us. And it is not about exactly the food but the probiotics in it. In 2012 our team published an article about the connection of probiotics and glucose metabolism (1). At the same time, the whole scientific world worked on different relationships between the gut microbiome and body functions. For literally few years ago, there is a massive progress of knowing the fine mechanisms and importance on this condition – the symbiosis with ``the good`` bacteria. If we think about the reason why it is so popular today and why so many researchers work on it, we only can point out the modern lifestyle and the bad quality of food accompanied it. The relationship between those two is the busy lifestyle and the necessary to eat the canned meal fulfilled of preservatives and poor of microbiome, combined with often antibiotic uses which destroyed your balance in long-term plan. Those states of continuing do`s eventually can ruin your life through the nervous system. We can conclude that the misdiagnose of dysbiosis is the 21st century challenge.
Stroke is an important cerebrovascular disease resulting in long-term disability and death. A rare consequence of stroke is structural epilepsy. Here we discuss the importance of post-stroke epilepsy prevention. We look into the occurrence of stroke and epilepsy in the general population, the risk factors and severity of those conditions and the significance of early seizure prevention after the stroke. We analyze published research papers and reviews dealing with this problem. In brief, there is a diagnostic problem leading to ineffective treatment. On one hand preventing seizures in patients with no history of epilepsy, treating them with medications with known side effects. On the other hand by not treating them, risk seizures and future repercussions for their health and wellbeing. Here we propose ways to select patients for treatment and refine the diagnostic considerations for early start of anti-seizure therapy.
Cerebral toxoplasmosis is a common opportunistic infection that causes expansive brain lesions in people living with HIV/AIDS. But it is extremely rarely associated with HIV negative patients. This study presents a case of a 23-year-old male with non HIV-relative cerebral toxoplasmosis. There was an acute onset of the disease. The first symptoms included fever and signs of meningoradicular irritation. Neurological abnormalities progressed in cranial nerves disorders and pyramidal syndrome. The cerebrospinal fluid (CSF) changes were consistent with those of viral meningoencephalitis. CT and MRI showed multiple ring-enhancing lesions with high-intensity signals in cerebrum brain stem and cerebellum. The tests for HIV (ELISA and Western blot) were negative. Serological detection of Toxoplasma gondii (T. gondii) IgM and IgG antibodies in the CSF confirmed the diagnosis. Parasitic, bacterial, viral encephalitis and meningoencephalitis, as well as neoplastic metastatic process and other neurological diseases were discussed in the differential diagnosis. The intensity of the disease activity fluctuated during the hospital stay regardless of the adequate therapy.
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