[1] We report on laboratory experiments which investigate interactions between aseismic slip, stress changes, and seismicity on a critically stressed fault during the nucleation of stick-slip instability. We monitor quasi-static and dynamic changes in local shear stress and fault slip with arrays of gages deployed along a simulated strike-slip fault (2 m long and 0.4 m deep) in a saw cut sample of Sierra White granite. With 14 piezoelectric sensors, we simultaneously monitor seismic signals produced during the nucleation phase and subsequent dynamic rupture. We observe localized aseismic fault slip in an approximately meter-sized zone in the center of the fault, while the ends of the fault remain locked. Clusters of high-frequency foreshocks (M w~À 6.5 to À5.0) can occur in this slowly slipping zone 5-50 ms prior to the initiation of dynamic rupture; their occurrence appears to be dependent on the rate at which local shear stress is applied to the fault. The meter-sized nucleation zone is generally consistent with theoretical estimates, but source radii of the foreshocks (2 to 70 mm) are 1 to 2 orders of magnitude smaller than the theoretical minimum length scale over which earthquake nucleation can occur. We propose that frictional stability and the transition between seismic and aseismic slip are modulated by local stressing rate and that fault sections, which would typically slip aseismically, may radiate seismic waves if they are rapidly stressed. Fault behavior of this type may provide physical insight into the mechanics of foreshocks, tremor, repeating earthquake sequences, and a minimum earthquake source dimension.
Direct shear sliding experiments on bare ground surfaces of Westerly granite have been conducted over an exceptionally wide range of sliding rates (10−4 µm/s to 10³ µm/s) at unconfined normal stresses (σn) of 5, 15, 30, 70, and 150 MPa. A new sample configuration was developed that permitted measurements at normal stresses of 70 and 150 MPa without immediate sample failure. Measurements of steady‐state velocity dependence of friction at velocities between 10−4 and 1 µm/s show similar velocity weakening behavior at all normal stresses, with more negative dependence at lower slip rates. However, at rates above 10 µm/s, velocity weakening is observed only at σn=30, 70 and 150 MPa, while velocity neutral behavior is observed at σn=15 MPa and velocity strengthening is observed at σn=5 MPa. The greater velocity weakening observed at velocities below 10−2 µm/s may suggest a transition in competing deformation mechanisms, or the influence of additional mechanisms. The transition to velocity strengthening at high velocity and low normal stress implies that rapid slip on shallow faults could be arrested before resulting in true stick‐slip behavior. Stable fault creep and creep events observed at shallow levels on some natural faults may result from this transition in velocity dependence.
Earthquake instability occurs as a result of strength loss during sliding on a fault. It has been known for over 50 years that fault compaction or dilatancy may cause significant weakening or strengthening by dramatically changing the fluid pressure trapped in faults. Despite this fundamental importance, we have no real understanding of the exact conditions that lead to compaction or dilation during nucleation or rupture. To date, no direct measurements of pore pressure changes during slip in hydraulically isolated faults have been reported. We show direct examples of fluid pressure variations during nucleation and rupture using a miniature pressure transducer embedded in an experimental fault. We demonstrate that fluids not only are significant in controlling fault behavior but can provide the dominant mechanism controlling fault stability. The effect of fluid pressure changes can exceed frictional variations predicted by rate‐ and state‐dependent friction laws, exerting fundamental controls on earthquake rupture initiation.
Changes in fault normal stress can either inhibit or promote rupture propagation, depending on the fault geometry and on how fault shear strength varies in response to the normal stress change. A better understanding of this dependence will lead to improved earthquake simulation techniques, and ultimately, improved earthquake hazard mitigation efforts. We present the results of new laboratory experiments investigating the effects of step changes in fault normal stress on the fault shear strength during sliding, using bare Westerly granite samples, with roughened sliding surfaces, in a double direct shear apparatus. Previous experimental studies examining the shear strength following a step change in the normal stress produce contradictory results: a set of double direct shear experiments indicates that the shear strength of a fault responds immediately, and then is followed by a prolonged slip-dependent response, while a set of shock loading experiments indicates that there is no immediate component, and the response is purely gradual and slip-dependent. In our new, high-resolution experiments, we obsen'e that the acoustic transmissivity and dilatancy of simulated faults in our tests respond immediately to changes in the normal stress, consistent with the interpretations of previous investigations, and verify an immediate increase in the area of contact between the roughened sliding surfaces as normal stress increases. However, the shear strength of the fault does not immediately increase, indicating that the new area of contact between the rough fault swfaces does not appear preloaded with any shear resistance or strength. Additional slip is required for the fault to achieve a new shear strength appropriate for its new loading conditions, consistent with previous observations made during shock loading.
scite is a Brooklyn-based organization that helps researchers better discover and understand research articles through Smart Citations–citations that display the context of the citation and describe whether the article provides supporting or contrasting evidence. scite is used by students and researchers from around the world and is funded in part by the National Science Foundation and the National Institute on Drug Abuse of the National Institutes of Health.