Human-Object Interaction Detection tackles the problem of joint localization and classification of human object interactions. Existing HOI transformers either adopt a single decoder for triplet prediction, or utilize two parallel decoders to detect individual objects and interactions separately, and compose triplets by a matching process. In contrast, we decouple the triplet prediction into human-object pair detection and interaction classification. Our main motivation is that detecting the human-object instances and classifying interactions accurately needs to learn representations that focus on different regions. To this end, we present Disentangled Transformer, where both encoder and decoder are disentangled to facilitate learning of two subtasks. To associate the predictions of disentangled decoders, we first generate a unified representation for HOI triplets with a base decoder, and then utilize it as input feature of each disentangled decoder. Extensive experiments show that our method outperforms prior work on two public HOI benchmarks by a sizeable margin. Code will be available.
Plant shoot branching is pivotal for developmental plasticity and crop yield. The formation of branch meristems is regulated by several key transcription factors including REGULATOR OF AXILLARY MERISTEMS1 (RAX1), RAX2, and RAX3. However, the regulatory network of shoot branching is still largely unknown. Here, we report the identification of EXCESSIVE BRANCHES1 (EXB1), which affects axillary meristem (AM) initiation and bud activity. Overexpression of EXB1 in the gain-offunction mutant exb1-D leads to severe bushy and dwarf phenotypes, which result from excessive AM initiation and elevated bud activities. EXB1 encodes the WRKY transcription factor WRKY71, which has demonstrated transactivation activities. Disruption of WRKY71/EXB1 by chimeric repressor silencing technology leads to fewer branches, indicating that EXB1 plays important roles in the control of shoot branching. We demonstrate that EXB1 controls AM initiation by positively regulating the transcription of RAX1, RAX2, and RAX3. Disruption of the RAX genes partially rescues the branching phenotype caused by EXB1 overexpression. We further show that EXB1 also regulates auxin homeostasis in control of shoot branching. Our data demonstrate that EXB1 plays pivotal roles in shoot branching by regulating both transcription of RAX genes and auxin pathways.
We propose a novel combination of a radiation field model and the transfer matrix method (TMM) to demonstrate plasmon-induced transparency (PIT) in bright-dark mode waveguide structures. This radiation field model is more effective and convenient for describing direct coupling in bright-dark mode resonators, and is promoted to describe transmission spectra and scattering parameters quantitatively in infinite element structures by combining it with the TMM. We verify the correctness of this novel combined method through numerical simulation of the metal-dielectric-metal (MDM) waveguide side-coupled with typical bright-dark mode, H-shaped resonators; the large group index can be achieved in these periodic H-shaped resonators. These results may provide a guideline for the control of light in highly integrated optical circuits.
scite is a Brooklyn-based organization that helps researchers better discover and understand research articles through Smart Citations–citations that display the context of the citation and describe whether the article provides supporting or contrasting evidence. scite is used by students and researchers from around the world and is funded in part by the National Science Foundation and the National Institute on Drug Abuse of the National Institutes of Health.