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The current study examined the accuracy of the multiple-stimulus without replacement (MSWO) preference assessment for identifying preferred common classroom activities as reinforcers with children with behavioral disorders. The accuracy of predictions from the MSWO regarding high, medium, and low stimulus preference was tested by providing contingent access to activities for completing math problems within an independent seatwork format. Overall, there was an interaction effect between preference ranking (high, medium, or low) and number of problems completed. The results confirm and extend previous findings regarding the accuracy of predictions with the MSWO. The findings also reveal, however, some individual differences that may account for instances in which student behavior did not conform to predictions of stimulus preference assessments.

Intellectual disability may appear to many as a barrier to participation in or the production of educational research. Indeed, a common perception of individuals seen as having cognitive impairments, and especially those with minimal or no verbal communication, is that they are incapable of the reasoning or lack the deliberative capacities necessary to participate in research or policy-influencing decision making. In this essay, Ashley Taylor dismantles these assumptions, challenging both the view of intellectual disability on which they rest and the view of epistemic competence they imply. Taylor shows how the absence or exclusion of people with intellectual disabilities labels from dominant knowledge-making institutions and arenas, including within educational research, amounts to injustice and results in their tacit or overt exclusion from civic education and political membership.

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<p>A growing body of work in feminist philosophy of disability, in particular, and philosophy of cognitive disability, more generally, demonstrates the discursive constitution of norms of intelligence and cognitive ability in order to undermine both the false assumptions about human ability and the gendered and racialized norms of rationality, independence, and competence that inform philosophical and bioethical theories about moral status. Many of these philosophical accounts of disability seem designed to&mdash;implicitly or explicitly&mdash;prove that, given some newly-valued norms, &nbsp;certain persons do indeed have these capabilities, rather than to transform the social conditions that create such demarcations in the first place. In this paper, I argue that feminist philosophy of disability and moral philosophy more broadly would benefit if they were to consider the social conditions of possibility in which these qualifications for moral status arise, rather than continue to focus on the qualifications themselves. In order to argue in this way, I consider how assessments of moral status and human life simultaneously foreclose possible expressions of "lives worth living." I suggest, furthermore, that feminist philosophers of disability in particular and feminist philosophers in general would benefit if they were to consider the risks that this normative theorizing involves. In turn, I propose a way in which feminist philosophers ought to orient themselves in order to create the conditions of possibility for the emergence of divergent expressions of human well-being and moral potential.&nbsp;</p><p>Keywords: personhood; intellectual disability; social justice; normative violence; feminist philosophy</p>

The purpose of this exploratory study was to investigate the extent to which a partnership orientation in conjoint behavioral consultation (CBC) may predict case outcomes and determine the relationship between a partnership orientation and implementation integrity of CBC. CBC is a problem-solving process by which families and teachers work collaboratively with a consultant to address students' academic, behavioral, and social needs. Twenty children, their parents and teachers, and consultants were involved in CBC services. Outcomes included parent and teacher ratings of acceptability, satisfaction, perceptions of effectiveness, and child performance across home and school settings. Partnership orientation scores and process integrity data were collected across interviews for each case. Results suggest that both parents and teachers consistently find CBC to be an acceptable, effective, and satisfactory form of service delivery. Findings reveal that consultants can conduct CBC interview objectives effectively within a partnership orientation. A partnership orientation in CBC was significant in predicting teachers' acceptability and satisfaction with the process; however, parents' acceptability and satisfaction with the CBC process was not predicted by a partnership orientation.

The growing field of feminist disability studies explores how human bodies are interpreted through cultural values and expectations surrounding physical and mental ability. This paper contributes to and expands upon this conversation by examining how the ideal of “able‐mindedness” functions to maintain racial divisions and inequalities through attributions of cognitive and psychiatric disability to bodies of color. Drawing upon contemporary examples from popular social media, public policy, and academic discourse, the author shows how racialized and nonnormatively gendered bodies are identified and interpreted through norms of able‐mindedness and used as markers against which the ideal of the able mind is upheld. This “discourse of pathology” operates insidiously within academic theorizing by remaining largely invisible because it tracks our deeply ingrained assumptions about the undesirability of cognitive and psychiatric disability. The author argues that because of the entanglement of race with disability, so long as the normalizing and privileging of the ideal of able‐mindedness goes unchallenged and we maintain the myth that there exists a normal mental state, both racism and ableism remain very much alive, including within the academy.

Serum Response Factor (SRF) is a ubiquitously expressed transcription factor and master regulator of the actin cytoskeleton. We have previously shown, that SRF is essential for megakaryocyte maturation and platelet formation and function. Here we elucidate the role of SRF in neutrophils, the primary defense against infections. To study the effect of loss of SRF in neutrophils, we crossed Srffl/fl mice with select Cre-expressing mice and studied neutrophil function in vitro and in vivo. Despite normal neutrophil numbers, neutrophil function is severely impaired in mice in whom Srf is selectively deleted in the hematopoietic system (Srf KO). Srf KO neutrophils fail to migrate to sites of inflammation in vivo. In a model mimicking lung infection by nebulization of lipopolysaccharide (LPS), significantly fewer neutrophils are recruited to the inflamed lungs 4 and 24 hours after LPS administration as evident by cell numbers retrieved in the bronchoalveolar lavage fluid (BAL, total cells: WT 0.568 ± 0.093x106 vs. KO 0.128 ± 0.024x106 at 4 hours and WT 1.337 ± 0.369x106 vs. KO 0.347 ± 0.045x106 at 24 hours; p <0.005 at 4 hours, p < 0.05 at 24 hours). Similarly, in an in vivo peritonitis model, where all other immune cells normally express Srf, significantly fewer Srf KO than WT neutrophils are recruited to the inflamed peritoneal space resulting in significantly reduced Srf KO neutrophil numbers in the peritoneal lavage fluid. To directly assess neutrophil migration and chemotaxis we performed in vitro transwell assays and assessed neutrophil migration in the Dunn chamber assay. Srf KO neutrophils show a significant migration defect in response to fMLP and KC. We next assessed actin polymerization in Srf WT and KO neutrophils. Srf KO neutrophils fail to polymerize globular actin to filamentous actin in response to fMLP. Neutrophil polarization in response to cytokine stimuli is markedly decreased. In addition, Srf KO neutrophils show markedly reduced adhesion. Integrins play an essential role in neutrophil adhesion and migration. Srf KO neutrophils show normal expression of the integrin LFA1 (CD11a/CD18), however, Mac1 (CD11b/CD18) expression is markedly increased on the cell surface as assessed by flow cytometry and Immunofluorescent staining, despite reduced mRNA expression. We find that trafficking of Mac1, essential for directed cell migration, is disrupted in Srf KO neutrophils, likely contributing to the migratory defect. Migration and cellular adhesion are essential for normal cell function, but also for malignant processes such as metastasis, thus underscoring an essential function for SRF and its pathway in health and disease. Disclosures: No relevant conflicts of interest to declare.