Many prograded barriers and some dunefields in the world have been termed 'beach ridge' plains, but the actual genesis of the 'ridges' is often unknown. Use of the terms, berms, beach ridges and foredunes is also confusing in the literature because their definitions are highly variable and are commonly used interchangeably. Thus, the formation and definition of sand berms, beach ridges and foredunes is briefly reviewed. Beach ridges are re-defined as entirely wave formed deposits which are most commonly formed during high wave conditions and/or elevated water levels (e.g. storm surges). Foredunes are formed by aeolian sand deposition in vegetation on the backshore. Some dunefields in Brazil have been called beach ridge plains when they are, in fact, foredune plains, transgressive dunefields, or complex barriers (i.e. barriers comprising two types of dunes). The Holocene barrier extending from Torres to Tramandaí in southern Brazil has been regarded as a beach ridge plain. The landforms of this Holocene barrier comprise wide, relatively linear, widely spaced (400-600m), shore parallel ridges on the landward half, and more closely spaced (80-400m), lobate and crescentic, discrete ridges on the seaward half. Low, rolling dunefields, sand sheets, nebkha fields and deflation plains occur between the ridges. The barrier is re-interpreted as a prograded, transgressive dunefield barrier.
It has been shown that emotional stress may induce oxidative damage, and considerably change the balance between pro-oxidant and antioxidant factors in the brain. The aim of this study was to verify the effect of repeated restraint stress (RRS; 1 h/day during 40 days) on several parameters of oxidative stress in the hippocampus of adult Wistar rats. We evaluated the lipid peroxide levels (assessed by TBARS levels), the production of free radicals (evaluated by the DCF test), the total radical-trapping potential (TRAP) and the total antioxidant reactivity (TAR) levels, and antioxidant enzyme activities (SOD, GPx and CAT) in hippocampus of rats. The results showed that RRS induced an increase in TBARS levels and in GPx activity, while TAR was reduced. We concluded that RRS induces oxidative stress in the rat hippocampus, and that these alterations may contribute to the deleterious effects observed after prolonged stress.
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