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In this paper, the unsteady motion of Casson liquid over a half‐infinite penetrable vertical plate with MHD, thermal radiation, Soret, and Dufour contributions have been explored numerically. In the physical geometry, the Casson liquid flows to the layer from the penetrable vertical plate. At the layer, Casson liquid is set into motion and the flow equations are illustrated using coupled partial differential equations (PDEs). This set of PDEs is simplified to form dimensionless PDEs with the use of normal nondimensional transformation. The controlling parameters' effects on the working fluid are extensively discussed on velocity, concentration, and temperature and presented graphically. Computational values of Nusselt plus Sherwood number and skin friction for controlling parameters are depicted in a tabular form. Our outcomes show that a raise in the Casson term depreciates the velocity because of the magnetic parameter influence on the fluid flow. The Soret parameter was found to accelerate the skin friction along with the Sherwood number coefficients. An incremental value of the Dufour parameter was detected to hike the skin friction alongside the Nusselt number. Results of this study were found to be in conformity with previously published work.

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The impact of heat and mass transfer effects on an MHD flow past an inclined porous plate in the presence of a chemical reaction is investigated in this study. An effort has been made to explain the Soret effect and the influence of an angle of inclination on the flow field, in the presence of the heat source, chemical reaction and thermal radiation. The momentum, energy and concentration equations are derived as coupled second order partial differential equations. The model is non-dimensionalized and shown to be controlled by a number of dimensionless parameters. The resulting dimensionless partial differential equations can be solved by using a closed analytical method. Numerical results for pertaining parameters, such as the Soret number (Sr), Grashof number (Gr) for heat and mass transfer, the Schmidt number (Sc), Prandtl number (Pr), chemical reaction parameter (Kr), permeability parameter (K), magnetic parameter (M), skin friction (τ), Nusselt number (Nu) and Sherwood number (Sh) on the velocity, temperature and concentration profiles are presented graphically and discussed qualitatively.

Our study tilt toward the deficiency of serum folic acid levels in diabetes mellitus patient with nephropathy. So if we would correct folic acid deficiency in diabetic patient then we could prevent the development of various complication associated with diabetes and help in reducing the morbidity and mortality of diabetic patient.

Thrombocytopenia is one of the most common hematological manifestations seen in HIV patients with approximately 40% of the patients developing thrombocytopenia during their course of illness. Opportunistic infection like tuberculosis is a rare but curative cause of thrombocytopenia in these patients. Clinically, it is a challenge to determine the exact cause and decide the treatment of thrombocytopenia in these patients as both infections can lead to significant thrombocytopenia by varied mechanisms. The treatment options are limited by drug antitubercular treatment highly active antiretroviral therapy (ATT and HAART)-induced thrombocytopenia. A 25-year-old male patient presented with low grade, intermittent fever with left-sided pleuritic chest pain for 2 months along with dry cough, weight loss, malaise, and lethargy. Hematological examination showed anemia, leukopenia, and thrombocytopenia. The patient was found to be HIV reactive. Chest X-ray revealed nonhomogenous opacity with cavitation in the left upper and middle zone suggestive of tuberculosis. He received multiple units of blood and platelets and was started on isoniazid, rifampicin, pyrazinamide, and ethambutol. HAART was initiated 2 weeks after starting ATT. Patient's hematological parameters improved and he was discharged on ATT and HAART with recovery of the platelet counts. Thrombocytopenia is a complication associated with both HIV and tuberculosis and hence determining the cause and deciding the appropriate treatment is challenging. Decision about whom and when to treat has to be taken cautiously considering all possible contributors and treatment has to be individualized.

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