“…In an earlier study, we showed that in a rat model of periodontal disease, basal mucin release from submandibular gland was increased [10]. The aim of the present work was to further study the mechanism underlying mucin secretion during experimental periodontitis with special focus on the relationship between the inflammatory mediators, such as, nitric oxide, prostaglandins and leukotrienes with the increase in mucin release observed in submandibular glands from rats with experimental periodontitis.…”