Venous Air Embolism

Campkin, T.V.; Perks, J

doi:10.1016/s0140-6736(73)90071-8

Cited by 10 publications

(3 citation statements)

References 3 publications

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“…4,9 Certainly, craniotomy performed in the sitting or semisitting position, as in the present case, increases the risk of intravenous air aspiration. 3 Our case is novel in that air aspiration appeared to occur at a low enough level that it did not cause changes in transthoracic Doppler sounds or hemodynamic instability. Rather, the best intraoperative evidence for ongoing aspiration included transient decreases in end-tidal CO 2 and an increasing requirement for a high inspired percentage of oxygen in the setting of observed aspiration of irrigant into cut bone surfaces.…”

Section: Discussionmentioning

confidence: 76%

Acute pulmonary edema after low-level air embolism during craniotomy

Frim

Wollman²,

Evans³

et al. 1996

Journal of Neurosurgery

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Acute pulmonary edema after a large air embolus occurring during neurosurgery is a recognized phenomenon. The authors describe the course of a 76-year-old man who presented with noncardiogenic pulmonary edema shortly after undergoing resection of a high convexity meningioma. Transthoracic Doppler sonography, however, showed no evidence of a large intraoperative emboli; the evidence for ongoing but low-magnitude air embolus included visualization of bone aspiration of irrigant before bone-edge waxing, transient intraoperative declines in end-tidal CO2 tension, and an increase of the fraction of inspired oxygen to maintain adequate saturation after removal of the craniotomy flap. There was no hemodynamic instability noted. The airspace disease was self-limited and resolved on supportive treatment after approximately 1 week, as would be expected for pulmonary edema caused by a single large intravenous air embolus. The authors present this case as the first report of pulmonary edema resulting from low-level air embolus occurring during craniotomy. This situation may go unrecognized intraoperatively but can cause the same significant postoperative morbidity as larger, more easily identified air emboli.

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Section: Discussionmentioning

confidence: 76%

Acute pulmonary edema after low-level air embolism during craniotomy

Frim

Wollman²,

Evans³

et al. 1996

Journal of Neurosurgery

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“…Por outro lado, o diagnóstico de embolia venosa pode ser realizado pelo ecocardiograma transesofágico, doppler precordial, cateterização da artéria pulmonar, medição do end-tidal de dióxido de carbono e saturação do oxigênio (Quadro 3) [31][32][33][34][35] . A hipertensão e a hipotensão são prejudiciais para o tratamento dessa entidade.…”

Section: Embolia Gasosa Venosaunclassified

Embolia gasosa

Locali¹,

Almeida²

2006

Rev. bras. ter. intensiva

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JUSTIFICATIVA E OBJETIVOS: A embolia gasosa é uma lesão iatrogênica que possui alta morbimortalidade. É uma complicação decorrente de procedimentos clínico-cirúrgicos, sendo necessário o conhecimento dessa entidade pelos profissionais de saúde. O objetivo deste estudo foi realizar uma revisão dessa entidade, abrangendo seus aspectos fisiopatológicos, diagnósticos e terapêuticos. CONTEÚDO: Este artigo revisa conceitualmente a embolia gasosa, separando-a em arterial e venosa. Relata as principais características fisiopatológicas, diagnósticas e terapêuticas das embolias arterial e venosa. Além disso, aborda-se a embolia paradoxal, evento advindo da conversão de embolia venosa em arterial. CONCLUSÕES: A embolia gasosa é uma complicação relevante e presente em muitas especialidades médicas. Uma compreensão acerca da sua fisiopatologia e dos métodos diagnósticos e terapêuticos é essencial para garantir maior segurança aos pacientes. Unitermos: Embolia, embolia aérea, embolia paradoxal SUMMARY BACKGROUND AND OBJECTIVES: Gas embolism is an iatrogenic injury that has high morbidity and morta

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“…Presumably, the subplacental venous sinuses are the site of air entry when antepartum or peripartum air embolism occurs [45]. More commonly, venous air embolism occurs during head and neck surgery, neurosurgical procedures, and in association with central venous catheter placement [47,50].…”

Section: Causes Of Acute Respiratory Failurementioning

confidence: 99%

Acute Respiratory Failure in Pregnancy

Hollingsworth¹,

Pratter²,

Irwin³

1989

J Intensive Care Med

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Acute respiratory failure in pregnancy has multiple etiologies, including thromboembolism, amniotic fluid embolism, venous air embolism, aspiration of gastric contents, respiratory infections, asthma, beta-adrenergic tocolytic therapy, and pneumomediastinum and pneu mothorax. Proper management of acute respiratory fail ure in pregnancy requires an understanding of the specific diseases and the normal gestational changes that occur in maternal respiration (decreased functional re sidual capacity, increased minute ventilation, mild respi ratory alkalosis) and hemodynamics (increased cardiac output, increased blood and plasma volume, unchanged central pressures). Knowledge of the determinants of oxygen delivery to fetal tissue (uterine blood flow, pla cental transfer, fetal circulation) and how they are af fected by changes in maternal hemodynamics, position, acid-base status, and medications can help sustain nor mal fetal development, whenever possible, without compromising maternal care. Diagnostic testing such as radiography, hemodynamic monitoring, and fetal moni toring are considered in terms of attendant risk to the mother or the fetus, alterations in normal values related to gestation, and indications for usage. Similarly, the risks and benefits of supportive and specific therapies for the various etiologies of acute respiratory therapy are reviewed.

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Venous Air Embolism

Cited by 10 publications

References 3 publications

Acute pulmonary edema after low-level air embolism during craniotomy

Acute pulmonary edema after low-level air embolism during craniotomy

Embolia gasosa

Acute Respiratory Failure in Pregnancy

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