Vascular Injuries After Percutaneous Transluminal Angioplasty

Bergentz, Sven‐Erik; Bergqvist, David

doi:10.1007/978-3-642-74086-2_5

Cited by 3 publications

(2 citation statements)

References 60 publications

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“…This complication is often attributed to faulty technique, consequent to puncture of the external iliac artery above the inguinal ligament, or to puncture of the femoral artery distal to the bifurcation where tamponade may be ineffectual because of the lack of firm underlying tissue on which postprocedural compression relies for hemostasis. 3,12 Arteriovenous fistula formation was seen with surprising frequency in this study period. A likely explanation is the simultaneous puncture of the artery for coronary dilation and the adjacent vein for right heart access and pacing, although the Seldinger technique itself may be causative.…”

Section: Discussionmentioning

confidence: 51%

Postcatheterization vascular complications associated with percutaneous transluminal coronary angioplasty

Oweida¹,

Roubin²,

Smith³

et al. 1990

Journal of Vascular Surgery

206

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Section: Discussionmentioning

confidence: 51%

Postcatheterization vascular complications associated with percutaneous transluminal coronary angioplasty

Oweida¹,

Roubin²,

Smith³

et al. 1990

Journal of Vascular Surgery

206

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“…To prevent thrombosis after PTA, medication with vari ous drugs including antiplatelet agents has been advocat ed (13). Aspirin, a cyclooxygenase inhibitor, is a potent inhibitor of platelet aggregation and is frequently prescribed for patients who underwent PTA so as to pre vent abrupt reocclusion.…”

mentioning

confidence: 99%

Effects of a Thromboxane A2-Receptor Antagonist, a Thromboxane Synthetase Inhibitor and Aspirin on Prostaglandin I2 Production in Endothelium-Intact and -Injured Aorta of Guinea Pigs

Higo

Karasawa

1994

Japanese Journal of Pharmacology

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ABSTRACT-We examined the effects of KW-3635, a thromboxane (TX) A2-receptor antagonist, and OKY-046, a TX synthetase inhibitor, on the prostaglandin (PG) 12 production in endothelium-intact and -in jured guinea pig aorta and compared them with those of aspirin. In the endothelium-intact aorta, both the low (3 mg/kg) and the high (100 mg/kg) dose of aspirin similarly reduced the PGI2 production, as measured ex vivo 1 hr after the injury. In contrast, neither KW-3635 (10 mg/kg) nor OKY-046 (30 mg/kg) inhibited the PGI2 production. The endothelial injury, induced by balloon catheterization, caused a reduction of PGI2 production in the aorta and decline of plasma PGI2/TXA2 ratio. In the endothelium-injured animals, the high dose of aspirin further reduced the PGI2 production in the aorta, whereas KW-3635 and OKY-046 did not affect it. KW-3635 and OKY-046 also ameliorated the reduced ratio of PGI2/TXA2 in the plasma. The present results demonstrate that aspirin, but not KW-3635 or OKY-046, reduces the PGI2 production in the aorta either in the endothelium-intact or -injured state. It is thus suggested that the TXA2-receptor an tagonist and the TX synthetase inhibitor have some advantages over aspirin when used for the prevention of acute thrombosis after percutaneous transluminal angioplasty.

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Vessel repair after balloon angioplasty: Morphological appearance and prostacyclin synthesising capacity

Mattsson

Brunkwall

Fält

et al. 1992

European Journal of Vascular Surgery

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Vascular Injuries After Percutaneous Transluminal Angioplasty

Cited by 3 publications

References 60 publications

Postcatheterization vascular complications associated with percutaneous transluminal coronary angioplasty

Postcatheterization vascular complications associated with percutaneous transluminal coronary angioplasty

Effects of a Thromboxane A2-Receptor Antagonist, a Thromboxane Synthetase Inhibitor and Aspirin on Prostaglandin I2 Production in Endothelium-Intact and -Injured Aorta of Guinea Pigs

Vessel repair after balloon angioplasty: Morphological appearance and prostacyclin synthesising capacity

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