Background and Purpose: Acute paraplegia must be investigated promptly to exclude reversible causes. In this report we illustrate the usefulness of transesophageal echocardiography in identifying the vascular etiologies of acute paraplegia.Case Descriptions: Two patients presented with acute paraplegia, one spontaneously and the other after removal of an intra-aortic balloon pump catheter. Through the use of transesophageal echocardiography, we excluded aortic dissection and identified protruding atherosclerotic plaques in the descending thoracic aorta of each patient. Embolization of atheromatous material from the thoracic aorta was considered the most likely etiology of paraplegia in both cases.Conclusions: Embolization from atherosclerotic plaques in the thoracic aorta may be an underestimated cause of acute paraplegia. Transesophageal echocardiography provides a safe, rapid, and reliable tool for investigating a vascular etiology of acute paraplegia. T he causes of acute paraplegia include malignant metastases to the spinal cord, trauma, aortic dissection, and spinal artery occlusion from thrombosis and embolism. Identification of the etiologic mechanism of acute paraplegia is not difficult when dealing with trauma or malignancies; for example, a magnetic resonance imaging (MRI) scan can quickly identify a compressive lesion. However, identifying a vascular etiology can be more challenging. Angiography has been the standard diagnostic test when a vascular cause, especially an aortic dissection, is suspected. However, less-invasive techniques such as transesophageal echocardiography (TEE) have been shown to be as reliable as angiography. In this report, we describe two patients with acute paraplegia in whom TEE was useful for demonstrating the probable etiology for the acute paraplegia.
Case Reports Case 1A 77-year-old woman with a history of hypertension and coronary artery disease was admitted to a local hospital with sudden onset of epigastric pain radiating to her back, followed by paraplegia. Aortic dissection was suspected, and an emergency computed tomographic (CT) scan was performed and interpreted as normal. Upon transfer to our institution, her neurological examination was significant for sensory deficits at the T12 level on the left and at the T4 level on the right, with areflexia and marked motor weakness of her lower extremities.