Urinary Oxalate Excretion and its Role in Vulvar Pain Syndrome

Baggish, Michael S.; Sze, Ehm; Johnson, R.A.

doi:10.1097/00128360-199804000-00023

Cited by 15 publications

(21 citation statements)

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“…The use of anticonvulsants, such as gabapentin, have been promoted for treatment of vulvodynia . A combination of low‐oxalate diet and oral calcium citrate has been proposed as a treatment of hyperoxaluria and mycroscopic oxalcrystaluria when a deficiency is considered one of the etiopathogenic causes of the refractory vestibulodynia …”

Section: Medical Treatmentmentioning

confidence: 99%

Vulvodynia—An Evidence‐Based Literature Review and Proposed Treatment Algorithm

et al. 2015

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The optimal therapy for vulvar pain syndrome remains elusive, with low percentages of therapeutic success, using either local or systemic pharmacological approaches. Surgery involving invasive and often irreversible therapeutic procedures has resulted in success for certain subtypes of vulvodynia. We present a multidisciplinary approach whereby pain treatment units may provide an intermediate level of care between standard medical and surgical treatments.

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Section: Medical Treatmentmentioning

confidence: 99%

Vulvodynia—An Evidence‐Based Literature Review and Proposed Treatment Algorithm

et al. 2015

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“…The woman experienced a cessation of her bladder pain symptoms [57]. Another study compared urinary oxalate excretion in women with vulvodynia vs controls, and evaluated the role of dietary intervention in women with vulvodynia [58]. The results showed no difference in the 24‐h excretion of oxalate between the two groups.…”

Section: Comorbidities Of Ic/bps and Dietmentioning

confidence: 99%

Diet and its role in interstitial cystitis/bladder pain syndrome (IC/BPS) and comorbid conditions

2012

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What ' s known on the subject? and What does the study add? Nearly 90% of patients with interstitial cystitis/bladder pain syndrome (IC/BPS) report sensitivities to a wide variety of dietary comestibles. Current questionnaire-based literature suggests that citrus fruits, tomatoes, vitamin C, artifi cial sweeteners, coffee, tea, carbonated and alcoholic beverages, and spicy foods tend to exacerbate symptoms, while calcium glycerophosphate and sodium bicarbonate tend to improve symptoms. At present we recommend employing a controlled method to determine dietary sensitivities, such as an elimination diet, in order to identify sensitivities while at the same time maintain optimal nutritional intake.We review current literature with regard to diet ' s effect upon IC/BPS and common comorbidities (irritable bowel syndrome, fi bromyalgia, chronic fatigue syndrome, neuropathic pain, vulvodynia, and headache) with a focus upon questionnaire-based investigations. We discuss the pathologic mechanisms that may link diet and IC/BPS related-pain, concentrating upon specifi c comestibles such as acidic foods, foods high in potassium, caffeine, and alcohol.

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“…The true etiology is still a subject of debate, and recent reviews on the subject say more about what it is not than what it is (38–41). Some of the theories include chronic infection with Candida, herpes, and human papillomavirus (HPV), contact dermatitis, urticaria, hyperoxaluria, and premature use of oral contraceptives (42–48). However, the evidence is mostly anecdotal, and in the case of HPV, has been strongly queried (2,49).…”

Section: Managementmentioning

confidence: 99%

Management of vulvar pain

Fischer

2004

Dermatol Ther

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Vulvodynia is a frequently used medical term that literally means "vulvar pain". Therefore, vulvodynia is a symptom, not a disease. The term itself indicates a variety of unpleasant chronic vulvar sensations, including burning, rawness, soreness, irritation, sensitivity, and formication. This may or may not include dyspareunia. Primary vulvodynia occurs when these sensory disturbances occur in the absence of observable dermatologic disease or vulvovaginal infection. There are several causes for this, including neuropathy, referred pain, and pelvic floor muscle dysfunction. For the purist, it is the patient in whom there is no observable reason for vulvar pain who represents the true case of vulvodynia. However, vulvodynia can also occur secondarily as a symptom of vulvar skin disease. Restricting the present paper to patients without objective signs leaves out all the important conditions which come into the differential diagnosis of vulvar pain which should be ruled out first. The first step in managing vulvodynia is making an accurate diagnosis of its cause. The present review summarizes the diagnosis and management of the chronic dermatologic diseases which may cause primary and secondary vulvodynia. The etiology of primary vulvodynia is much more poorly understood than secondary vulvodynia, and treatment of some aspects remains controversial.

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Urinary Oxalate Excretion and its Role in Vulvar Pain Syndrome

Cited by 15 publications

References 0 publications

Vulvodynia—An Evidence‐Based Literature Review and Proposed Treatment Algorithm

Vulvodynia—An Evidence‐Based Literature Review and Proposed Treatment Algorithm

Diet and its role in interstitial cystitis/bladder pain syndrome (IC/BPS) and comorbid conditions

Management of vulvar pain

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