Uremic Pericarditis and Cardiac Tamponade in Chronic Renal Failure

Beaudry, C

doi:10.7326/0003-4819-64-5-990

Cited by 84 publications

(32 citation statements)

References 14 publications

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“…In addition, since the amount of fluid needed to cause tamponade is usually in excess of a liter, much more fluid can be removed in this manner than by a safe tap. In this case two previous attempts, using large bore needles, Circulation, Volume XXXVIII, September 1968 failed to give more than temporary relief.…”

Section: Commentmentioning

confidence: 92%

Uremic Pericarditis

et al. 1968

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SUMMARYUremic pericarditis occurred in 41% of 83 patients admitted to the chronic dialysis program at the Peter Bent Brigham Hospital. In the vast majority of these patients the pericarditis was present before dialysis and cleared clinically after beginning therapy. Instances of pericarditis that developed during regular dialysis were associated with metabolic stress such as surgery or infection or inadequate dialysis. When the pericarditis failed to resolve the patients usually died with sepsis or had severe tamponade which necessitated early pericardiectomy. The two cases reported illustrate resorption of massive pericardial effusions with dialysis. The utility of percutaneous pericardial catheterization as a treatment for uremic pericardial tamponade is demonstrated.

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Section: Commentmentioning

confidence: 92%

Uremic Pericarditis

et al. 1968

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“…Indeed, no case of bleeding due to hemodialysis-associated thrombocytopenia has been described. However, reports of spontaneous bleed ing episodes in patients on hemodialysis maintenance therapy frequently fail to report the patients' platelet counts, either pre-or post-dialysis [1][2][3][4][5][6][7][8][9], suggesting that some cases of bleeding due to hemodialysis-associated thrombocytopenia may have gone unrecognized. There fore, the clinical significance of hemodialysis-associated thrombocytopenia may be underestimated.…”

Section: Introductionmentioning

confidence: 99%

“…The increased incidence of spontaneous bleeding epi sodes observed in patients undergoing maintenance he modialysis therapy [1][2][3][4][5][6][7][8][9][10] suggests a defect of hemostasis in these patients. Potential causes of this defect are ure mic platelet dysfunction not entirely corrected by hemo dialysis, heparin anticoagulation during hemodialysis, and warfarin anticoagulation sometimes used to main tain vascular shunt patency.…”

Section: Introductionmentioning

confidence: 99%

Massive Hemorrhage due to Hemodialysis-Associated Thrombocytopenia

1983

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Marked declines in platelet numbers were noted in association with hemodialysis. This resulted in gastrointestinal bleeding and the need for packed RBC and platelet transfusions. This hemodialysis-associated thrombocytopenia was ameliorated by changing the dialyzer in use. The gastrointestinal bleeding stopped and the need for platelet transfusions was obviated. The degree of platelet loss during hemodialysis is probably affected by the composition of the dialyzer membrane used as well as other factors. Hemodialysis-associated thrombocytopenia may be a contributing factor in the increased bleeding tendency noted in hemodialysis patients.

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“…Pericarditis and pericardial effusion are common and serious complications of uremia [4,9,11,21,30,42], They develop in the terminal phase of renal insufficiency demanding immediate and violent hemodialysis or, if not successful, pericardiocentesis or pericardiectomy [14. 21], Moreover, renal transplantation may prevent peri carditis [20], Pericarditis also occurs in patients on regu lar hemodialysis [1,2,11,42] despite excellent biochem ical control and it has been assumed that this is a distinct form of pericarditis.…”

Section: Introductionmentioning

confidence: 99%

Humoral Immune Reactions in Uremic Pericarditis

Maisch¹,

Kochsiek²

1983

Am J Nephrol

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Clinical data from 41 patients with pericarditis or pericardial effusion in acute and chronic renal failure were analyzed in respect to the diagnostic relevance of humoral immune reactions. In patients with pericardial effusion in acute renal failure due to surgery or trauma (n = 10), antimyocardial antibodies were rarely detected. In contrast, in the sera of all patients with pericarditis or pericardial effusion following renal failure in chronic or acute glomerulonephritis (n = 6), complement-fixing antisarcolemmal antibodies of the IgM and IgG classes were observed. In patients in whom pericarditis or pericardial effusion evolved during chronic hemodialysis (n = 25), the incidence of complement-fixing antimyolemmal antibodies was 64%. Only sera with complement-fixing antimyolemmal antibodies induced cytolysis of vital adult cardiocytes in vitro suggesting that the antimyolemmal antibodies may not only play a diagnostic but also a pathogenetic role in ‘uremic’ pericarditis in vivo.

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Uremic Pericarditis and Cardiac Tamponade in Chronic Renal Failure

Cited by 84 publications

References 14 publications

Uremic Pericarditis

Uremic Pericarditis

Massive Hemorrhage due to Hemodialysis-Associated Thrombocytopenia

Humoral Immune Reactions in Uremic Pericarditis

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