Trombose da veia de Galeno: relato de caso

Marques, Marcio Chaves; Pires, Leopoldo Antônio; Damasceno, Carlos Augusto de Albuquerque; Felı́cio, André Carvalho; Atala, Ângelo; Franco, G

doi:10.1590/s0004-282x2003000200025

Cited by 4 publications

(3 citation statements)

References 18 publications

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“…Van den Bergh et al [ 4 ] published a series of 4 cases with partial syndromes with good clinical results. Case reports with bilateral thalamic ischemia have shown variable neurological outcome [ 7 , 8 ]. Crawford et al [ 6 ] retrospectively described 7 cases of thrombosis of the deep system with 5 deaths.…”

Section: Discussionmentioning

confidence: 99%

Thrombosis of the Vein of Galen: Pitfalls, Metamorphosis, and Paroxysmal Sympathetic Hyperactivity

Santos-Soares

Oliveira‐Filho

2017

Case Rep Neurol

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Isolated thrombosis of the vein of Galen is a rare and serious entity with few cases reported in the literature. We report the case of a previously healthy 18-year-old male who was admitted after developing headache and subsequently worsening mental status, requiring endotracheal intubation for airway protection. During his admission he developed symptoms of severe paroxysmal sympathetic hyperactivity and posturing. The computed tomography and magnetic resonance imaging of the brain showed bilateral thalamic lesions. The magnetic resonance angiography and digital arteriography revealed a thrombosis of the deep cerebral venous system (vein of Galen). We call attention to a case with rapid symptom progression and specific radiological findings, with atypical clinical course, characterized by paroxysmal sympathetic hyperactivity, but with good clinical functional outcome.

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Section: Discussionmentioning

confidence: 99%

Thrombosis of the Vein of Galen: Pitfalls, Metamorphosis, and Paroxysmal Sympathetic Hyperactivity

Santos-Soares

Oliveira‐Filho

2017

Case Rep Neurol

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“…In approximately 85% of cases, the thrombosis affects either the superior sagittal sinus or the transverse sinus (8)(9)(10) . Among the imaging methods, transfontanellar ultrasonography (TFUS) associated with Doppler fluxometry is the initial evaluation method, particularly because of the wide availability and bed-side capability, with low cost, and non-necessity of sedation or ionizing radiation exposure, but magnetic resonance imaging (MRI) remains as the gold standard for the diagnosis of CVT (5,6,9,11,12) .…”

Section: Introductionmentioning

confidence: 99%

Diagnóstico por imagem e aspectos clínicos da trombose venosa cerebral em recém-natos a termo sem dano cerebral: revisão em 10 anos

Monteiro

Lima

Ribeiro³

et al. 2010

Radiol Bras

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OBJECTIVE: To describe and compare imaging methods and clinical findings of cerebral venous thrombosis in four full-term neonates without brain damage, admitted to a neonatal intensive care unit. MATERIALS AND METHODS: Ten-year review of four cases diagnosed with cerebral venous thrombosis by transfontanellar ultrasonography associated with Doppler fluxometry and confirmed by magnetic resonance imaging/magnetic resonance angiography in correlation with clinical findings and neurological progression. RESULTS: Ultrasonography presented normal results in 75% of cases and magnetic resonance imaging in 100%. Doppler fluxometry and magnetic resonance angiography were abnormal in 100% of cases. Hypoxia (100%) and early seizures (100%) were predominant among clinical findings with evoked potential changes in 50% of cases. In the assessment of the neurodevelopment all the areas remained within normality parameters up to the conclusion of the present study. CONCLUSION: Ultrasonography in association with Doppler can identify changes related to cerebral venous thrombosis and should be complemented with magnetic resonance imaging that is the gold standard for diagnosis in these cases. Keywords: Cerebral venous thrombosis; Full-term neonates; Magnetic resonance imaging; Magnetic resonance angiography; Ultrasonography and Doppler.OBJETIVO: Descrever e comparar os métodos de imagem e os aspectos clínicos em quatro recém-natos a termo diagnosticados como trombose venosa cerebral, sem dano encefálico, adscritos a uma unidade de terapia intensiva neonatal. MATERIAIS E MÉTODOS: Revisão em 10 anos com quatro casos diagnosticados como trombose venosa cerebral por meio de ultrassonografia transfontanela com Doppler e confirmados por ressonância magnética/angiorressonância, correlacionados aos aspectos clínicos e evolução neurológica. RESULTADOS: A ultrassonografia foi normal em 75% dos casos e a ressonância magnética, em 100%. No caso alterado, a dilatação venosa foi identificada. O Doppler e a angiorressonância estavam alterados em 100% dos casos. Dos aspectos clínicos, a hipóxia (100%) e a convulsão precoce (100%) predominaram, com potencial evocado alterado em 50% dos casos. Na avaliação do neurodesenvolvimento, todas as áreas estiveram dentro da normalidade até a última avaliação. CONCLUSÃO: A ultrassonografia associada ao Doppler é capaz de identificar as alterações da trombose venosa cerebral, devendo ser complementada com a ressonância magnética, que é o padrão ouro de diagnóstico. Unitermos: Trombose venosa cerebral; Recém-nascidos a termo; Imagem por ressonância magnética; Angiorressonância; Ultrassonografia e Doppler. AbstractResumo * Study developed at Casa de Saúde São José (CSSJ-RJ), Rio de Janeiro, RJ, Brazil.

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“…There is also a third group without specific etiology, which may c o rrespond to 20% to 35% of the total causes 4 . C h ronic hepatitis C virus (HCV) infection has been c o n s i d e red as a rare cause of cerebral venous thro mbosis [5][6][7][8][9][10] .…”

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Cerebral venous thrombosis and hepatitis: case report

Felı́cio

Fukujima

Fiorot

et al. 2006

Arq. Neuro-Psiquiatr.

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-Among the many infective causes of cerebral venous thrombosis (CVT), viral hepatitis is been re g a rded as a rare associated condition. We re p o rt on a 56-years-old man presenting CVT associated with hepatitis B and C coinfections outlining probable pathogenic mechanisms. We suggest that virus B and C serology should be performed in the cases of cerebral venous thrombosis with unknown etiology.KEY WORDS: cerebral venous thrombosis, hepatitis B virus, hepatitis C virus.Trombose venosa cerebral e hepatite: relato de caso RESUMO -Dentre as várias causas infecciosas de trombose venosa cerebral (TVC), a hepatite viral tem sido reconhecida como causa rara de TVC. Relatamos sobre um homem de 56 anos com TVC associada a coinfecção pelos vírus B e C da hepatite, ressaltando possíveis mecanismos patogênicos. Sugerimos que soro l ogia para vírus da hepatite B e C deveria ser solicitado em todos os casos de TVC de origem indeterm i n a d a . PALAVRAS-CHAVE: trombose venosa cerebral, vírus da hepatite B, vírus da hepatite C.The main recognized causes or predisposing conditions to cerebral venous thrombosis (CVT) can be divided into two groups: infectious (either local or systemic) and non-infectious causes 1 -3 . There is also a third group without specific etiology, which may c o rrespond to 20% to 35% of the total causes 4 . C h ronic hepatitis C virus (HCV) infection has been c o n s i d e red as a rare cause of cerebral venous thro mbosis 5-10 . We re p o rt a case of such association and discuss possible physiopathogenic mechanisms. CASEA 56-year-old white man was admitted to our hospital with a 15-day history of headache worsening with considerable change in its frequency and intensity patterns. He was a chronic daily headache suff e rer taking amitry p t i l i n e 75 mg and propranolol 80 mg per day and had already taken several abortive treatment regimens. Four days before admission he had two episodes of focal motor seizure. His past history included heavy smoking and drinking. He took intravenous cocaine for 20 years until 12 years ago. He also had occasional bisexual relations. General physical examination was unremarkable. Neurological exam abnorm a l ities included left mild hemiparesis. Pain relief medication was initiated as well as carbamazepine 600 mg per day.Routine blood and urine exams were unremarkable. On c e rebral spinal fluid (CSF) the initial pre s s u re was 33 cm/H 2 O with cell count and biochemistry within normal parameters.Chest radiograph was normal such as cranial computed tomography scan (CT). Cranial magnetic resonance imaging (MRI) indicated superior sagittal and left transverse sinuses thrombosis associated to corpus callosum and right parietal hemorrhagic venous infarcts. New blood sample was taken in order to investigate pos sible secondary e tiologies and on that time venous non-fractionated heparin was initiated.Values of serum immunoglobulins were: IgA=151 mg/dL (78-367 mg/dL), IgG=703 mg/dL (583-1761 mg/dL) and IgM= 69 mg/dL (52-335 mg/dL). The following autoantibodi...

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Trombose da veia de Galeno: relato de caso

Cited by 4 publications

References 18 publications

Thrombosis of the Vein of Galen: Pitfalls, Metamorphosis, and Paroxysmal Sympathetic Hyperactivity

Thrombosis of the Vein of Galen: Pitfalls, Metamorphosis, and Paroxysmal Sympathetic Hyperactivity

Diagnóstico por imagem e aspectos clínicos da trombose venosa cerebral em recém-natos a termo sem dano cerebral: revisão em 10 anos

Cerebral venous thrombosis and hepatitis: case report

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