1966
DOI: 10.1016/0002-9149(66)90439-5
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Traumatic ventricular septal defects

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Cited by 15 publications
(3 citation statements)
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“…The first postulates that the rupture occurs due to acute compression of the heart between the sternum and the vertebrae during the late diastolic phase when the ventricles are filled and the atrioventricular valves are closed4). The second proposes that myocardial injury causes a microvascular disruption, leading to infarction and liquefaction of the septum5). In our case, the patient showed normal cardiac structure at the time of injury, but after 8 days, he was diagnosed as having a VSD, after repeated echocardiography.…”
Section: Discussionmentioning
confidence: 99%
“…The first postulates that the rupture occurs due to acute compression of the heart between the sternum and the vertebrae during the late diastolic phase when the ventricles are filled and the atrioventricular valves are closed4). The second proposes that myocardial injury causes a microvascular disruption, leading to infarction and liquefaction of the septum5). In our case, the patient showed normal cardiac structure at the time of injury, but after 8 days, he was diagnosed as having a VSD, after repeated echocardiography.…”
Section: Discussionmentioning
confidence: 99%
“…Early mechanical rupture may occur when a sudden high pressure tears the ventricular septum at the end-diastole when the ventricles are filled and valves are closed [5]. Delayed inflammatory rupture may be caused by defective microcirculation associated with myocardial contusion, leading to necrosis and subsequent rupture of ventricular septum [6]. It is possible that early mechanical rupture causes sudden hemodynamic overload, whereas delayed inflammatory rupture causes gradual hemodynamic changes, leading to more frequent decompensation in patients diagnosed in the early phase.…”
Section: Case Presentationmentioning
confidence: 99%
“…The initial damage of the nutrient vessels of the septum or papillary muscles results in infarction which progresses to liquefaction necrosis and perforation or rupture (Williams and Bullock, 1961). 2.…”
Section: Pathology Of Traumatic Cardiac Lesions Thementioning
confidence: 99%