Transplacental naphthalene poisoning

Anziulewicz, John A.; Dick, Herman J.; Chiarulli, Eugene E.

doi:10.1016/0002-9378(59)90522-8

Cited by 31 publications

(8 citation statements)

References 5 publications

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“…It is well recognized that naphthalene haemolytic anaemia is by no means rare, particularly following the ingestion of mothballs by children, and that glucose-6-phosphate dehydrogenase deficiency predisposes to haemolysis (Dacie, 1967) Transplacental poisoning with haemolytic anaemia has been reported in a newborn infant whose mother sucked mothballs during the last trimester of pregnancy (Anziulewicz et al, 1959).…”

Section: Discussionmentioning

confidence: 99%

Toxic Haemolytic Anaemia in Pregnancy Due to a Pica for Paradichlorobenzene

Campbell¹,

Davidson

1970

BJOG

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Section: Discussionmentioning

confidence: 99%

Toxic Haemolytic Anaemia in Pregnancy Due to a Pica for Paradichlorobenzene

Campbell¹,

Davidson

1970

BJOG

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“…During pregnancy, reduced fetal birth weight and growth are more frequently observed on PAH exposure (Duarte-Salles et al 2013;Perera et al 2005;WHO 2010a, b). The developing fetus remains on cancer risk, due to susceptibility to DNA damage (Perera et al 2005) and hemolytic anemia after trans-placental transport exposure to PAHs (Zinkharn and Childs 1958;Anziulewicz et al 1959). In children, exposure to PAHs can induce oxidative stress (Svecova et al 2009;Li et al 2013;Kamal et al 2015), genotoxicity (EC and European Commission 2001; Gamboa et al 2008;Kim et al 2013), neurological dysfunction (Saunders et al 2002;Kim et al 2013), hepato-toxicity and hematological changes, including anemia IARC 1983;Kamal et al 2014a, b;Kim et al 2013 and reference therein).…”

Section: Health Risks Associated With Pahs Exposurementioning

confidence: 95%

A review of PAH exposure from the combustion of biomass fuel and their less surveyed effect on the blood parameters

Kamal

Cincinelli

Martellini

et al. 2014

Environ Sci Pollut Res

117

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Many epidemiological studies from all over the world have reported that populations of rural and urban environments differ in their health issues due to the differences in the countrywide pollution pattern. In developing countries, various occupational cohorts and subsections of the population in urban and rural areas are routinely exposed to several environmentally widespread contaminants. Polycyclic aromatic hydrocarbons (PAHs) are a group of over hundred different compounds and have ubiquitous presence in rural and urban environments. Smoke from the combustion of biomass fuel contains a high concentration of carcinogenic PAHs, which are related with several human morbidities. The sources and types of biomass fuel are diverse and wide in distribution. Limited numbers of literature reports have focused the significant impact of PAHs on several components of blood, both in human and wildlife. The toxicity of PAHs to rapidly dividing cells (e.g., bone marrow cells) and other tissues is largely attributed to their reactive oxygenated metabolites, potential of causing oxidative stress, and the adducts of their metabolites with DNA. This review aims to encompass the blood-related effects of PAHs and associated human health risks-an aspect that needs further research-on the population of developing countries of the world in particular.

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“…Naphthalene can cross the placenta and is fetotoxic (79). In 21 newborns, a rather severe form of hemolytic anemia was produced by blankets stored in naphthalene (35).…”

Section: 424mentioning

confidence: 99%

Polycyclic Aromatic Hydrocarbons and Azaaromatic Compounds

Baxter¹,

Warshawsky²

2012

Patty's Toxicology

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Polycyclic aromatic hydrocarbons (PAHs) are moderately reactive, but undergo photochemical degradation in the atmosphere, and are widely used as chemical raw materials. Aromatic hydrocarbons cause local irritation and changes in endothelial cell permeability and are absorbed rapidly. Accumulation of aromatic hydrocarbons in marine animals occurs to a greater extent and retention is longer compared to alkanes. Toxicity of polynuclear aromatics has been reported comprehensively. It has been reported that exposure to a variety of complex mixtures containing these chemicals, such as soot, coal tar and pitch, mineral oils, coal gasification residues, and cigarette smoke has historically been associated with induction of cancer. Naphthalene causes cataracts in the eyes of experimental animals. Its vapors may cause severe systemic injury. Alkylbenzenes are readily aspirated and produce instant death via cardiac arrest and respiratory paralysis. In general, the acute toxicity of alkylbenzenes is higher for toluene than that for benzene and decreases further with increasing chain length of the substituent, except for highly branched C 8 to C 18 derivatives. Polycyclic aromatic hydrocarbons are metabolized through epoxides and hydroxides and are excreted as conjugates.

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Transplacental naphthalene poisoning

Cited by 31 publications

References 5 publications

Toxic Haemolytic Anaemia in Pregnancy Due to a Pica for Paradichlorobenzene

Toxic Haemolytic Anaemia in Pregnancy Due to a Pica for Paradichlorobenzene

A review of PAH exposure from the combustion of biomass fuel and their less surveyed effect on the blood parameters

Polycyclic Aromatic Hydrocarbons and Azaaromatic Compounds

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