1983
DOI: 10.1378/chest.84.5.638
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Transient Left Posterior Hemiblock during Prinzmetal's Angina Culminating in Acute Myocardial Infarction

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Cited by 5 publications
(6 citation statements)
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“…Furthermore, patients with CVS may present with transient intraventricular conduction disorders such as bifascicular block, left anterior or posterior hemiblock and right bundle branch block, although this is rare. [ 6 ] CVS is also associated with various arrhythmias such as sinus bradycardia, sinus arrest with or without junctional escape beats, ventricular tachycardia, ventricular fibrillation and asystole. [ 3 ] Right coronary artery vasospasms often cause bradyarryhthmias as in Case 1, while left anterior descending artery vasospasms are prone to tachyarrhythmias.…”
Section: Discussionmentioning
confidence: 99%
“…Furthermore, patients with CVS may present with transient intraventricular conduction disorders such as bifascicular block, left anterior or posterior hemiblock and right bundle branch block, although this is rare. [ 6 ] CVS is also associated with various arrhythmias such as sinus bradycardia, sinus arrest with or without junctional escape beats, ventricular tachycardia, ventricular fibrillation and asystole. [ 3 ] Right coronary artery vasospasms often cause bradyarryhthmias as in Case 1, while left anterior descending artery vasospasms are prone to tachyarrhythmias.…”
Section: Discussionmentioning
confidence: 99%
“…in acute inferior myocardial infarction was described by Ortega-Carnicer, Garcia-Nieto, Malillos, & Sanchez-Fernandez, (1983), acute anterolateral myocardial infarction (Ogawa, Kimura, Okada, Ogino, & Katayama, 1976) and induced by exercise in the setting of severe coronary artery disease (Bobba, Salerno, & Casari, 1972).…”
Section: (A) (B) (C) (D) (E) (F)mentioning
confidence: 97%
“…However, when we analyze the depth of the q wave in III, qIII in a > qIII in c, which indicates that the initial portions of ventricular activation are more significant in a than in c, consequently, a higher degree of LPFB in a in the initial phase and a higher degree in c in the midfinal phase. in acute inferior myocardial infarction was described by Ortega-Carnicer, Garcia-Nieto, Malillos, & Sanchez-Fernandez, (1983), acute anterolateral myocardial infarction (Ogawa, Kimura, Okada, Ogino, & Katayama, 1976) and induced by exercise in the setting of severe coronary artery disease (Bobba, Salerno, & Casari, 1972).…”
Section: Introductionmentioning
confidence: 97%
“…presented a case of a 49-year-old man with transient LPFB during Prinzmetal's angina with STE in the inferior leads; subsequently LPFB reappeared associated with acute inferior MI. These authors believe that the very early changes in the QRS axis and ST segment are explained by three important changes in leads overlying the affected area: 1) the conspicuous STE associated with reciprocal changes; 2) the “injury”-related intramural (local) block; and 3) the increase of the R-wave voltage, also dependent of dromotropic disturbance caused by the “injury” [19]. Note: This prominent R wave could explain the LSFB observed in the proximal obstruction of the LAD before its first septal perforator branch.…”
Section: Epidemiologymentioning
confidence: 99%