Tonic contractions of the tensor tympani muscle: a key to some non-specific middle ear symptoms? Hypothesis and data from temporal bone experiments

Pau, Hans Wilhelm; Punke, C.; Zehlicke, Thorsten; Dressler, Dirk; Sievert, Uwe

doi:10.1080/00016480510012408

Cited by 28 publications

(21 citation statements)

References 15 publications

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“…The contraction of the tensor tympani and stapedius muscles stabilizes the ossicular chain and has its tension vector in a mediocaudal plane [14]. When it is considered that when applying pressure on the scala vestibuli by pressing medially on the stapes, significant endolymphatic movement is caused (i.e.…”

Section: Discussionmentioning

confidence: 99%

Tenotomy of the middle ear muscles causes a dramatic reduction in vertigo attacks and improves audiological function in definite Meniere's disease

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Hamzavi

et al. 2011

Acta Oto-Laryngologica

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Section: Discussionmentioning

confidence: 99%

Tenotomy of the middle ear muscles causes a dramatic reduction in vertigo attacks and improves audiological function in definite Meniere's disease

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Hamzavi

et al. 2011

Acta Oto-Laryngologica

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“…Second, the TM is a unique structure suspended in the air [16]. This is also pronounced with the fact that sound transmission or changes in atmospheric pressure can cause the malleus handle and the tympanic myotonic contraction [17]. The eardrum flap has no tension, poor flexibility, and is easy to contract.…”

Section: Discussionmentioning

confidence: 99%

Effect of treatment at different time intervals for traumatic tympanic membrane perforation on the closure

Lou¹,

Hu²,

Tang³

2011

Acta Oto-Laryngologica

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In the group treated with the gelatin sponge patch technique, the patients sought medical treatment at different time intervals since the injury. Accordingly, the outcome of the treatment varied in terms of the perforation closure rates achieved in different patients in this group. The respective perforation closure rates were 100%, 100%, 96%, 94%, and 89% in accordance with the time interval at which the patients were treated since the injury. The results were not significantly different when compared by statistical analysis (p > 0.05); the mean closure times in each of the different sets of cases in this group were calculated and the following values were reported: 7.1 ± 2.3, 8.2 ± 1.6, 8.7 ± 1.2, 9.2 ± 3.1, and 10.7 ± 3.9 days. On the other hand, in the edge approximation plus gelfoam patching group, the perforation closure rates were 100%, 97%, 96%, 97%, and 94%, respectively. This was in accordance with the time elapsed since the injury for the patients who visited the hospital on different days. Statistical analysis confirmed that the perforation closure rates for the different cases of this group did not have any significant difference (p > 0.05); the mean closure times were 7.6 ± 1.9, 7.9 ± 2.2, 9.2 ± 2.8, 8.5 ± 3.6, and 11.2 ± 4.1 days, respectively, indicating that differences were not significant even in terms of mean closure rates for the different cases of this group (p > 0.05).

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“…These severe cellular injuries occur rapidly after alkaline ingestion, within minutes from the contact and even for small quantities or low concentration of alkaline solutions [7] , thus making it difficult to support a hypothesis of such a sectorial sufferance. However, a change in middle ear pressure, which is reported to be able to influence the function of the inner ear [8] , seems improbable to coexist with unaffected tubal function; for the same reason, functional anomalies of the tensor tympani, although credited as having some possible adverse effect on the labyrinth [9] , and considered in the hypthesized link between labyrinthine disorders and gastroesophageal reflux [10] , might have a hypothetic marginal role. Finally, the most probable explanation could be an indirect, systemic effect casually involving the inner ear as a sensory organ with a blood supply of terminal type.…”

Section: Discussionmentioning

confidence: 99%

Sudden Bilateral Sensorineural Hearing Loss as an Unusual Consequence of Accidental Ingestion of Potassium Hydroxide

Ciorba

Bovo²,

Castiglione³

et al. 2010

Med Princ Pract

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Objective: To discuss the possible etiopathogenetic mechanism of inner ear damage induced by the ingestion of potassium hydroxide (KOH). Clinical Presentation and Intervention: We report the case of a 37-year-old patient with sudden bilateral sensorineural hearing loss after accidental ingestion of a KOH solution. The first ear, nose and throat examination disclosed only mild edema of the upper airways. He was treated in the intensive care unit and prescribed high-dose steroids, proton pump inhibitors and sucralfate for 2 weeks. Unfortunately, there was no recovery of the hearing loss, and no audiogram changes were noticed after 12 months of follow-up.Conclusion: After exploring the possible etiopathogenetic mechanism involved, the authors believe that in this case, a transient severe hemodynamic imbalance can actually be considered to be the most reliable explanation for the inner ear damage and subsequent onset of permanent bilateral sensorineural hearing loss.

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Tonic contractions of the tensor tympani muscle: a key to some non-specific middle ear symptoms? Hypothesis and data from temporal bone experiments

Cited by 28 publications

References 15 publications

Tenotomy of the middle ear muscles causes a dramatic reduction in vertigo attacks and improves audiological function in definite Meniere's disease

Tenotomy of the middle ear muscles causes a dramatic reduction in vertigo attacks and improves audiological function in definite Meniere's disease

Effect of treatment at different time intervals for traumatic tympanic membrane perforation on the closure

Sudden Bilateral Sensorineural Hearing Loss as an Unusual Consequence of Accidental Ingestion of Potassium Hydroxide

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