Thyroid dysfunctions in morphoea: A preliminary report

Hassan, Iffat; Arif, Tasleem; Anwar, Parvaiz

doi:10.4103/0378-6323.144230

Cited by 6 publications

(6 citation statements)

References 4 publications

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“…The examination of 17 patients with different types of morphea revealed normal thyroid functions in two patients with linear morphoea ( 111 ), but another types of localized scleroderma were accompanied in some cases by thyroid diseases ( 43 , 112 ).…”

Section: Resultsmentioning

confidence: 99%

Progressive Hemifacial Atrophy and Linear Scleroderma En Coup de Sabre: A Spectrum of the Same Disease?

Хамаганова

2018

Front. Med.

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Similar clinical and histhopathological features in progressive hemifacial atrophy and linear scleroderma en coup de sabre are well known. Trauma may predispose to the development of both diseases. The lack of association with anti-Borrelia antibodies was shown in both cases as well. The otolaryngological and endocrine disorders may be associated findings in both diseases. However, there are certain differences in neurological and ophthalmological changes in the diseases.

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Section: Resultsmentioning

confidence: 99%

Progressive Hemifacial Atrophy and Linear Scleroderma En Coup de Sabre: A Spectrum of the Same Disease?

Хамаганова

2018

Front. Med.

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“…In some cases, it is not possible to determine the cause of the disease; in such a situation, the patient is managed as described in this paper. The literature contains reports on many disorders of the skin and subcutaneous tissue, which are described as typically coexisting with thyroid diseases 4 . Frisch et al 5 report on vitiligo and alopecia areata as the most typical disorders.…”

Section: Discussionmentioning

confidence: 99%

“…Morphea and lichen sclerosus are immune diseases that may be associated with other autoimmune disorders. The coexistence of two autoimmune diseases may not occur by chance, but could be attributed to autoimmune reactions elicited by recognition of common antigens [4][5][6][7] .…”

Section: Introductionmentioning

confidence: 99%

Morphea and Lichen Sclerosus in a Patient with Hyporthyroidism

Bergler‐Czop

Brzeźińska-Wcisło

2020

ACC

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Thyroid gland is one of the key organs regulating the metabolism of carbohydrates, proteins and fats. Its primary function is connected with increase of the metabolic conversion of the body. Skin lesions are often one of the first symptoms of hypothyroidism. In a 71-year-old patient, skin lesions in the form of thickened areas with severe hyperkeratosis first appeared 12 years before. Eight years before, the patient was diagnosed with hypothyroidism of unknown cause. Upon admission, lesions were observed in the trunk area, left arm and vulva. These lesions had the appearance of brownish spots with hyperkeratosis. Initially, they were localized on the trunk, then involving upper limbs and neck area with time. Significant progression was present in the back area. In the anogenital area, porcelain-white discolorations were observed. Laboratory examinations were normal. During hospital stay, iv. ceftriaxone at a dose of 2.0 g/day for 10 days and intramuscular injection of vitamin B6 were administered, along with 10% urea ointment for hyperkeratosis lesions as topical therapy. The patient had lesions in the form of skin induration and discoloration, with visible, very severe hyperkeratosis, which is not characteristic of changes of the scleroderma and lichen sclerosus type. These lesions caused diagnostic problems due to the atypical clinical appearance.

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“…It is well known that different types of scleroderma can follow pregnancy [9 -11] Hassan I & colleagues, (2014) examined thyroid dysfunctions in morphoea. The levels of thyroid stimulating hormone were elevated with 41.2% patients [12].…”

Section: Introductionmentioning

confidence: 99%

“…[11] thyroid dysfunctions Bonilla-Abadia F., Muñoz-Buitron E, Ochoa CD, Carrascal E., Cañas CA (2012) [13],Hassan I., Arif T, Anwar P.(2014) [12] diabetes mellitus Firoz EF, Kamino H, Lehman TJ, Orlow SJ(2010) [22] [63] vitamin B 12 Verdelli The Endocrine Changes…”

Section: Introductionmentioning

confidence: 99%

Localized Scleroderma: Predisposing and Triggering Factors

Хамаганова¹

2017

TODJ

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Abstract:The etiology of localized scleroderma remains unclear. The objective of this article is to review different theories of etiology, specifically provocative and predisposing factors for the development of localized scleroderma. The mini-review presents the factors, which may influence the occurrence of localized scleroderma. The genetic predisposition for the development of scleroderma is a relevant characteristic of the disease. Different traumatic factors, viral & bacterial infections, chemical substances, pharmaceutical agents may trigger localized scleroderma. Neoplasia is regarded as a distinct triggering impulse for scleroderma.

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Thyroid dysfunctions in morphoea: A preliminary report

Cited by 6 publications

References 4 publications

Progressive Hemifacial Atrophy and Linear Scleroderma En Coup de Sabre: A Spectrum of the Same Disease?

Progressive Hemifacial Atrophy and Linear Scleroderma En Coup de Sabre: A Spectrum of the Same Disease?

Morphea and Lichen Sclerosus in a Patient with Hyporthyroidism

Localized Scleroderma: Predisposing and Triggering Factors

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