“…P1 and P2 phenotypes of the P blood group system, excess of Rh negativity, excess of Lewis Lea-b-, excess of salivary ABH non-secretors, and excess of HLA A2, A3, but lack of HLA A10, B5, BW6, BW35 are the samples for these studies [8][9][10][11][12][13][14][15][16][17][18][19][20][21]. Additionally, the correlation of A/AB phenotypes with Staphylococcus Saprofiticus induced UTI, A phenotype with Pseudomonas Aeruginosa-induced otitis externa, and A phenotype with Schistosomia Haematobium infections were reported [22,23]. In a contradiction with these studies, Albarus et al [7] and Schaffer et al [24] could not confirmed the previous findings suggesting different susceptibility to UTI based on phenotypes of several genetic markers.…”