The mechanism of hypertension in the Guillain-Barré syndrome

Mitchell, Paul; Meilman, Edward

doi:10.1016/0002-9343(67)90079-4

Cited by 48 publications

(16 citation statements)

References 33 publications

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“…Neurologic symptoms may be the initial finding [12]. As seen in the present case, low levels of VMA may occur in cases of pheochromocytoma, with paroxysmal, rather than sustained, hypertension.…”

Section: Scussl Onsupporting

confidence: 62%

Malignant pheochromocytoma in childhood: Report of a case with familial neurofibromatosis

Quissel¹,

Mohammad²,

Bauer³

et al. 1979

Med. Pediatr. Oncol.

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Section: Scussl Onsupporting

confidence: 62%

Malignant pheochromocytoma in childhood: Report of a case with familial neurofibromatosis

Quissel¹,

Mohammad²,

Bauer³

et al. 1979

Med. Pediatr. Oncol.

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“…Occa sionally, autonom ic nerves are involved, in which case there may be urinary retention, postural hypotension, or hypertension. Hypertension may be found in the course of GBS in 33% o f patients [3], The pathophysiologic basis for hypertension in GBS is unclear and reports have variously implicated sympathetic overactivity [4,5], increased renin release [6], and baroreceptor dys function [7], Our patient showed transient hypertension in accord with an overactivity of the sympathetic ner vous system assessed by increased plasma and urinary catecholamines. On the other hand, it is well known that GBS may cause SIADH, though the pathophysiologic basis is also unclear.…”

Section: Discussionmentioning

confidence: 75%

Inappropriate Secretion of Antidiuretic Hormone and Transient Hypertension Associated with Guillain-Barré Syndrome

Kaneko¹,

Shioya²,

Yabuta

1989

Pediatr Neurosurg

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A rare case with Guillain-Barré syndrome complicated by inappropriate secretion of antidiuretic hormone (SIADH) and hypertension was reported. This case showed clinically mild symptoms without respiratory muscle paralysis and had a complete neurological recovery. It should, therefore, be considered that the Guillain-Barré syndrome may cause SIADH and hypertension independent of the severity of the disease.

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“…Hypertension has been reported by several authors. [7][8] Meilman8 found an increased concen¬ tration of circulating catecholamines in the blood and urine of a patient with the Guillain-Barré syndrome who had hypertension.…”

Section: Report Of a Casementioning

confidence: 99%

Cardiac Monitoring and Demand Pacemaker in Guillain-Barre Syndrome

Emmons¹,

Blume²,

DuShane³

1975

Archives of Neurology

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Life-threatening alterations of respiratory and cardiovascular functions may complicate the course of severe Guillain-Barre syndrome. Cardiac arrest, fore-warned by episodes of bradycardia or other arrhythmia, may occur despite adequately assisted respirations. A patient with Guillain-Barre syndrome required tracheostomy and ventilatory assistance. Continuous cardiac monitoring revealed that tracheal aspiration provoked an idioventricular rhythm of 40 beats per minute, which reverted to sinus rhythm after the procedure. To prevent cardiac arrest during the transition from idioventricular to sinus rhythm, a demand pacemaker was inserted into the right ventricle. Set to activate if the rate fell below 65 beats per minute, the pacemaker functioned both during and between tracheal aspiration procedures. Cardiac monitoring in severe cases of Guillain-Barre syndrome may detect potentially lethal arrhythmias that may then be avoided by a demand pacemaker.

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The mechanism of hypertension in the Guillain-Barré syndrome

Cited by 48 publications

References 33 publications

Malignant pheochromocytoma in childhood: Report of a case with familial neurofibromatosis

Malignant pheochromocytoma in childhood: Report of a case with familial neurofibromatosis

Inappropriate Secretion of Antidiuretic Hormone and Transient Hypertension Associated with Guillain-Barré Syndrome

Cardiac Monitoring and Demand Pacemaker in Guillain-Barre Syndrome

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