The hepatitis A virus infection

Association between hepatitis A virus and Helicobacter pylori in a developing country: The saga continues

“…It is possible that the seropositivity in the study population for H. pylori and HAV reflects the age‐specific seroprevalence of both infections rather than a true association. In a similar study, the prevalence of HAV and H. pylori paralleled, but this was attributed to high age‐related seroprevalence of both infections 37 . It appears that the same factors that affected the age‐specific prevalence of HAV antibodies, may also predict the age‐specific prevalence of H. pylori antibodies, but to a lesser extent.…”

The Battle for Survival: The Role of RNA Non-Canonical Tails in the Virus–Host Interaction

“…Approximately 240 million patients with Chronic Hepatitis B (CHB) are Hepatitis B surface antigen (HbsAg) positive, exposed to the risk of cirrhosis and hepatocellular carcinoma (HCC) [ 61 , 62 ]. Unlike HBV, HAV infection usually causes acute hepatitis, ranging in severity from mild to severe [ 63 , 64 ]. In rare cases, a weakened immune system can make hepatitis A infection deadly.…”

Non‐A, non‐B hepatitis. Serological, clinical, morphological and prognostic aspects

“…Adjustment of the assay to detect anti-HAV IgM in the first 3 -4 months only enables differentiation between actual and past HAV infection. The presence of anti-HAV IgM is evidence of acute HAV infection, while the absence of anti-HAV IgM excludes actual infection (12). A chronic carrier state of HAV probably does not exist and development of chronic liver disease after HAV infection is rare if it occurs at all (12).…”