The grief response in neuropathologies of speech and language

Tanner, Dennis C.; Gerstenberger, Dean L.

doi:10.1080/02687038808248889

Cited by 51 publications

(19 citation statements)

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“…A more in-depth account of personal and emotional adjustment follows, based primarily on the interview evidence from the people with aphasia. Tanner and Gerstenberger's (1988) application of the grief model proposed that adjustment to impairment such as aphasia involved the stages of denial, frustration, and depression, progressing to acceptance. Evidence from the narratives aligned partially to this model, but showed additional processes, specifically concerning adjustment to the ongoing loss associated with aphasia, including restricted independence, also relating to physical loss.…”

Section: Personal and Emotional Adjustmentmentioning

confidence: 99%

“…Tanner and Gerstenberger (1988) proposed that the grief response (a model initially developed for characterising bereavement; Kubler- Ross, 1969) be applied to neuropathologies of speech and language, including aphasia, to account for dimensions of loss within relationships, self and the environment. Gainotti's (1997) review of emotional, psychological and psychosocial problems in aphasia considered this grief model-a progression from denial, through feelings of anger and frustration, to what Tanner and Gestenberger (1988) term "depression" and, providing that the depression is not "pathological", finally to acceptance.…”

Section: Exploring Adjustment As Change Over Timementioning

confidence: 99%

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Adjustment processes in chronic aphasia after stroke: Exploring multiple perspectives in the context of a community-based intervention

Mumby

Whitworth

2013

Aphasiology

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Background: The impact of chronic aphasia following stroke on quality of life (QOL) is widely acknowledged, with improved QOL recognised as an important outcome in aphasia recovery and supported by emerging quantitative measures. One of the key constructs recognised as contributing to QOL in other chronic conditions is psychosocial adjustment, the mechanisms of which are little understood for the person with aphasia. Aims: This study addressed adjustment processes in aphasia by exploring multiple perspectives from people engaged in the Communication Hub for Aphasia in North Tyneside (CHANT), a two-year community intervention for long-term aphasia. The study aimed to explore the adjustment process over time in people with aphasia using thematic analysis of personal narratives derived from a combination of sources: semi-structured interviews with reflections on experiences, quantitative measures of change in QOL and self-assessments of change.Methods & Procedures: Three people with mild or moderate chronic aphasia and three people without aphasia involved in CHANT were recruited (a carer, a volunteer, and a local government employee) to participate in semi-structured interviews at two-to threemonth intervals over a 12-month period. A total of 28 semi-structured interviews were transcribed and analysed thematically by a small team using NVivo8 software. Narrative data were interpreted within the broader context of QOL measures and self-assessments of living with aphasia (Mumby & Whitworth, 2012). Outcomes & Results: Changes over time that reflected evidence of psychosocial adjustment from the multiple perspectives of the participants covered five core themes: Intervention type, Effectiveness, Barriers, Facilitators, and QOL. A model is proposed to encapsulate the barriers and facilitators that impacted on the process of adjustment and

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Section: Personal and Emotional Adjustmentmentioning

confidence: 99%

Section: Exploring Adjustment As Change Over Timementioning

confidence: 99%

Adjustment processes in chronic aphasia after stroke: Exploring multiple perspectives in the context of a community-based intervention

Mumby

Whitworth

2013

Aphasiology

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“…The relationship between post-stroke functional impairment and PSDS is poorly understood [36]. Some have argued that stroke patients may develop depressive symptoms as a psychological reaction to limited physical functioning [37], others speculated that depressive symptoms themselves produce functional impairment as a result of fatigue, hopelessness, and diminished motivation [38] and a third point of view is that physical disabilities can both be a cause or a consequence of depressive symptoms [39]. …”

Section: Discussionmentioning

confidence: 99%

Post-Stroke Depressive Symptoms Are Associated with Post-Stroke Characteristics

et al. 2009

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Background: Several studies have described the frequency and risk factors of post-stroke depressive symptoms (PSDS). However, most studies did not exclude patients with depressive symptoms shortly before stroke and paid little attention to prestroke risk factors of depression, including previous depressive episodes, white matter lesions, and brain atrophy. These are potential limitations to assess the true effect of stroke on the occurrence of depressive symptoms. Our aim was to investigate the prevalence and risk factors of PSDS with adjustments for the previously mentioned prestroke factors. Methods: 420 consecutive patients with an acute clinical symptomatic transient ischemic attack or cerebral infarction were eligible for enrolment in this study. The presence of PSDS was rated by the Hospital Anxiety and Depression Scale 6–8 weeks after stroke. The relation between (pre-) stroke factors and PSDS was assessed with multivariate regression analysis. Results: The prevalence of PSDS was 13% and did not differ between stroke subtype or first-ever/ever occurrence of stroke. Higher degree of post-stroke handicap was related to PSDS (OR = 5.39; 95% CI = 2.40–12.08) and more functional independence had a protective effect on PSDS (OR = 0.88; 95% CI = 0.77–1.00). Conclusions: This is the largest study that investigated the prevalence and risk factors of PSDS by carefully excluding patients with depressive symptoms shortly before stroke. PSDS were not related to lesion side or location, but to the degree of post-stroke handicap and functional independence. Early detection of PSDS and their risk factors might help to predict long-term outcome and could promote early interventions of (behavioral) rehabilitation treatment strategies.

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“…A review of the other confounding factors would help the reader to have a clearer frame for understanding this phenomenon. Some researchers have argued that patients with stroke become depressed because of the psychologic reaction to limited physical functioning, 26 whereas others argue that depression leads to functional impairment as a result of fatigue, hopelessness, and diminished motivation. 27 A third group suggests that this is a mutual relationship; physical impairment could be a cause and effect of PSD.…”

Section: Introductionmentioning

confidence: 99%