The emotional ear in stress

Horner, Kathleen C.

doi:10.1016/s0149-7634(03)00071-x

Cited by 81 publications

(52 citation statements)

References 146 publications

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“…It excludes somatic symptoms and avoids potential confounding by somatic symptoms. It consists of independent subscales for anxiety and depression with scores in each scale interpreted in ranges as normal (0-7), mild (8-10), moderate (11)(12)(13)(14) and severe (15)(16)(17)(18)(19)(20)(21). The scoring system adopted by Snaith RP is used in this study in which a score of 11 or higher is regarded as mood disorder in anxiety and depression scales and a score of 8-10 being just suggestive of a disorder 21 .…”

Section: Assessment Toolsmentioning

confidence: 99%

“…The relationship between tinnitus and these comorbidities is complex and poorly understood and the lack of coping capabilities can play a critical role in the clinical history of severe tinnitus sufferers 12 . Results available suggest that cortical plasticity is likely responsible for the chronic progression of tinnitus 13 . However, the relationship between tinnitus and emotional distress has been researched to involve a complex connection between inner ear sensitivity and neuroendocrinological, immune and toxic variations that are linked to the activation of emotional stress 14 .…”

Section: Introductionmentioning

confidence: 99%

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The impact of tinnitus on adult Nigerians: health related Quality of Life assessment of sufferers using the Hospital Anxiety and Depression Scale (HADS) and the RAND-36 item health survey 1.0 questionnaire

Adoga¹,

Kokong²,

Nimkur³

et al. 2015

The International Tinnitus Journal

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Section: Assessment Toolsmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

The impact of tinnitus on adult Nigerians: health related Quality of Life assessment of sufferers using the Hospital Anxiety and Depression Scale (HADS) and the RAND-36 item health survey 1.0 questionnaire

Adoga¹,

Kokong²,

Nimkur³

et al. 2015

The International Tinnitus Journal

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“…The neurophysiological tinnitus model by Jastreboff and Hazell (1993) also emphasizes the role of heightened arousal as well as dysfunctional attention and appraisal processes. Various studies have evidenced a link between tinnitus and stress with consistently heightened stress levels that are accompanied by an increased cortical arousal level (Alpini and Cesarani 2006;Hebert and Lupien 2007;Horner 2003).…”

Section: Introductionmentioning

confidence: 99%

Stability of Physiological Variables in Chronic Tinnitus Sufferers

Weise

Heinecke

Rief

2008

Appl Psychophysiol Biofeedback

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The etiological tinnitus models propose that suffering can be caused and aggravated by heightened physiological arousal. Therefore psychophysiological treatments are applied. Stability of the measured parameters is essential for the use of biofeedback as well as to permit the attribution of changes to the administered treatment. The aim of our study was to investigate the 3-month reproducibility of psychophysiological parameters in 60 tinnitus patients. Using a repeated-measures design, the activity of these parameters was assessed twice during various stress and relaxation trials. The results showed that the measurements of frontalis, masseter and trapezius muscles were stable, while for the sternocleidomastoid, the skin conductance level (SCL) and the skin temperature retest-stability could not be evidenced. For all parameters, test-retest stability was weak for the relative scores. In conclusion, our study has important implications for applied psychophysiology research: (1) the measurement of EMG assessed in a clinical sample is stable over a 3-month interval; (2) in contrast, the measurements of SCL and skin temperature as well as all relative scores are less stable; and (3) the stability of EMG-parameters in our sample gives first hints that physiological changes can be attributed to an administered biofeedback treatment but further research is required.

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“…Among these symptoms are stiVness and pain in neck and shoulder area, sensory deWcits, motor dysfunction, muscle fatigue, local cervical mechanical hyperalgesia, temporo-mandibular dysfunction, often associated with dizziness, tinnitus, intermittent headache, signs of autonomic dysfunction, cognitive dysfunctions such as memory and concentration disturbances and psychological distress, depression and anxiety, and sleep disturbances (for review and refs, see Gimse et al 1997;Eck et al 2001;Horner 2003;Bergholm et al 2004;Rodriquez et al 2004;Ferrari et al 2005;Sterling et al 2005). The Quebec task force classiWcation of WAD (Spitzer et al 1995) is based on the type and severity of signs and symptoms observed shortly after injury, rather than on the speciWc type of lesion produced.…”

Section: Introductionmentioning

confidence: 99%

Influence of sympathetic nervous system on sensorimotor function: whiplash associated disorders (WAD) as a model

2006

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There is increasing interest about the possible involvement of the sympathetic nervous system (SNS) in initiation and maintenance of chronic muscle pain syndromes of different aetiology. Epidemiological data show that stresses of different nature, e.g. work-related, psychosocial, etc., typically characterised by SNS activation, may be a co-factor in the development of the pain syndrome and/or negatively affect its time course. In spite of their clear traumatic origin, whiplash associated disorders (WAD) appear to share many common features with other chronic pain syndromes affecting the musculo-skeletal system. These features do not only include symptoms, like type of pain or sensory and motor dysfunctions, but possibly also some of the pathophysiological mechanisms that may concur to establish the chronic pain syndrome. This review focuses on WAD, particular emphasis being devoted to sensorimotor symptoms, and on the actions exerted by the sympathetic system at muscle level. Besides its well-known action on muscle blood flow, the SNS is able to affect the contractility of muscle fibres, to modulate the proprioceptive information arising from the muscle spindle receptors and, under certain conditions, to modulate nociceptive information. Furthermore, the activity of the SNS itself is in turn affected by muscle conditions, such as its current state of activity, fatigue and pain signals originating in the muscle. The possible involvement of the SNS in the development of WAD is discussed in light of the several positive feedback loops in which it is implicated.

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The emotional ear in stress

Cited by 81 publications

References 146 publications

The impact of tinnitus on adult Nigerians: health related Quality of Life assessment of sufferers using the Hospital Anxiety and Depression Scale (HADS) and the RAND-36 item health survey 1.0 questionnaire

The impact of tinnitus on adult Nigerians: health related Quality of Life assessment of sufferers using the Hospital Anxiety and Depression Scale (HADS) and the RAND-36 item health survey 1.0 questionnaire

Stability of Physiological Variables in Chronic Tinnitus Sufferers

Influence of sympathetic nervous system on sensorimotor function: whiplash associated disorders (WAD) as a model

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