The Effects of Isoflurane and Isoflurane-Nitrous Oxide Anesthesia on Brainstem Auditory Evoked Potentials in Humans

Manninen, Pirjo; Lam, Arthur M.; Nicholas, James F.

doi:10.1097/00132586-198510000-00021

Cited by 24 publications

(8 citation statements)

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“…Previous studies have demonstrated small, but reliable increases in BAER latencies with sedation. In studies with persons without developmental disabilities, increased BAER latencies have been revealed by within subject comparisons for waves P3 and P5, but not P1, when using either enfluorane, halothane, isoflurane, or pentobarbital for anesthesia [Dubois et al, 1982;Thornton et al, 1984;Manninen et al, 1985;Drummond et al, 1987], and also a smaller increase in the latency of wave P1 when using high doses of sodium thiopental [Drummond et al, 1985]. Sedation was used in the present study to suppress movement artifacts and, as dictated by clinical judgment, consisted of different agents for different patients, thereby complicating comparison of the present results with those of the previous studies.…”

Section: Discussionmentioning

confidence: 99%

Longer brainstem auditory evoked response latencies of individuals with fragile X syndrome related to sedation

et al. 1997

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Brainstem auditory evoked response latencies were studies in 75 males (13 with fragile X syndrome, 18 with mental retardation due to other causes, and 44 with no disability). Latency values were obtained for each ear for the positive deflections of waves I (P1), III (P3), and V (P5). Some individuals with mental retardation required sedation. Contrary to previous report, latencies obtained for individuals with fragile X did not differ from those obtained for persons without mental retardation. Persons receiving sedation, whether or not their retardation was due to fragile X, had longer latencies for wave P5 than persons who did not receive sedation. This effect of sedation may also explain the previously reported increased latencies for persons with fragile X.

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Section: Discussionmentioning

confidence: 99%

Longer brainstem auditory evoked response latencies of individuals with fragile X syndrome related to sedation

et al. 1997

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“…The poor temporal resolution of ABR causes inconsistent monitoring 15. Because of the poor signal-to-noise ratio and poor temporal resolution, ABR is susceptible to disruption by various intraoperative factors, including general anaesthesia,16 body temperature,17 dural opening, saline irrigation of surgical field, surgical microscope, high-speed drill and ultrasonic aspirator 18 19. The high incidence of unsatisfactory ABR recordings degrades the usefulness of intraoperative ABR monitoring 18 – 20.…”

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confidence: 99%

Hearing preservation and intraoperative auditory brainstem response and cochlear nerve compound action potential monitoring in the removal of small acoustic neurinoma via the retrosigmoid approach

Yamakami

Yoshinori

Saeki

et al. 2009

Journal of Neurology, Neurosurgery & Psychiatry

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The retrosigmoid approach using auditory monitoring for a small AN can accomplish total tumour removal with an excellent hearing preservation rate. CNAP provides reliable auditory monitoring more frequently than ABR, reflects the intraoperative auditory function almost in real-time, predicts postoperative hearing with excellent sensitivity and specificity, and is more useful for monitoring in the removal of small AN with hearing preservation.

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“…The propofol is known to increase ABR latencies whereas amplitudes are decreased. It has been shown that isofurane is responsible for an increase of ABR latencies whereas it does not modify the amplitudes 11,12)…”

Section: Discussionmentioning

confidence: 99%

Effect of Isoflurane on the Hearing in Mice

et al. 2012

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Background and ObjectivesThe aim of this study was to investigate the relationship between inhalation anesthetics and hearing in mice.Materials and MethodsAs inhalation anesthetics, isoflurane was used. Auditory brainstem response and distortion product otoacoustic emission were used as measurement of hearing. Mice were divided into 2 groups. 'Isoflurane group' consisted of mice that were anesthetized with an inspired concentration of 2.0 vol% isoflurane with 2 L/min of oxygen (n=10). 'Control group' consisted of mice that were anesthetized with ketamine and xylazine (n=10).ResultsAuditory brainstem response thresholds in mice anesthetized with ketamine and xylazine was not different from those in mice anesthetized with isoflurane. Threshold of DPOAE was higher in mice with isolurane than with ketamine and xylazine. Changes of efferent control may be induced by isoflurane and consequently change the threshold of DPOAE in mice.ConclusionsThese results infer that, there was a change of central nervous system induced by inhalation anesthetics, this change also can be applied to the strategies for prevention of hearing loss.

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The Effects of Isoflurane and Isoflurane-Nitrous Oxide Anesthesia on Brainstem Auditory Evoked Potentials in Humans

Cited by 24 publications

References 0 publications

Longer brainstem auditory evoked response latencies of individuals with fragile X syndrome related to sedation

Longer brainstem auditory evoked response latencies of individuals with fragile X syndrome related to sedation

Hearing preservation and intraoperative auditory brainstem response and cochlear nerve compound action potential monitoring in the removal of small acoustic neurinoma via the retrosigmoid approach

Effect of Isoflurane on the Hearing in Mice

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