The assessment of carotid–femoral distance for aortic pulse wave velocity: Should it be estimated from body height?☆

Filipovský, Jan; Mayer, Otto; Dolejšová, M.; Seidlerová, Jitka

doi:10.1016/j.artres.2010.01.002

Cited by 10 publications

(9 citation statements)

References 8 publications

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“…Weber et al 6 produced a calculation for distance being equal to the (height/4+7.28) from a population of 135 being investigated for coronary artery disease, but, however, used the distance from the ascending aorta to the aortic bifurcation as the comparator which is not representative of the path traveled by the pulse wave between the carotid and femoral artery. Filipovsky et al 18 derived the calculation (height×0.29) in a population of 596 individuals predominantly free from cardiovascular disease but used an externally measured distance as the gold standard with all the aforementioned weaknesses this entails. Both techniques were examined by Huybrechts et al 4 in a population of 98 healthy men and women, which found that the Weber calculation was accurate but less so than a direct distance with a modifier of 0.8 and the Filipovsky calculation to be highly inaccurate.…”

Section: Discussionmentioning

confidence: 99%

Development and Validation of a Path Length Calculation for Carotid–Femoral Pulse Wave Velocity Measurement

et al. 2018

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Section: Discussionmentioning

confidence: 99%

Development and Validation of a Path Length Calculation for Carotid–Femoral Pulse Wave Velocity Measurement

et al. 2018

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“…Additionally, aortic pulse wave velocity (PWV) was estimated using the time lag between aortic Pf and Pb (Qasem and Avolio, 2008). Carotid-femoral distance was estimated by multiplying body height by 0.29 which may reduce measurement bias due to body disproportion that can occur with the standard tape measure method (Filipovsky et al, 2010). …”

Section: Methodsmentioning

confidence: 99%

Effect of acute resistance exercise on carotid artery stiffness and cerebral blood flow pulsatility

2014

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Arterial stiffness is associated with cerebral flow pulsatility. Arterial stiffness increases following acute resistance exercise (RE). Whether this acute RE-induced vascular stiffening affects cerebral pulsatility remains unknown. Purpose: To investigate the effects of acute RE on common carotid artery (CCA) stiffness and cerebral blood flow velocity (CBFv) pulsatility. Methods: Eighteen healthy men (22 ± 1 yr; 23.7 ± 0.5 kg·m−2) underwent acute RE (5 sets, 5-RM bench press, 5 sets 10-RM bicep curls with 90 s rest intervals) or a time control condition (seated rest) in a randomized order. CCA stiffness (β-stiffness, Elastic Modulus (Ep)) and hemodynamics (pulsatility index, forward wave intensity, and reflected wave intensity) were assessed using a combination of Doppler ultrasound, wave intensity analysis and applanation tonometry at baseline and 3 times post-RE. CBFv pulsatility index was measured with transcranial Doppler at the middle cerebral artery (MCA). Results: CCA β-stiffness, Ep and CCA pulse pressure significantly increased post-RE and remained elevated throughout post-testing (p < 0.05). No changes in MCA or CCA pulsatility index were observed (p > 0.05). There were significant increases in forward wave intensity post-RE (p < 0.05) but not reflected wave intensity (p > 0.05). Conclusion: Although acute RE increases CCA stiffness and pressure pulsatility, it does not affect CCA or MCA flow pulsatility. Increases in pressure pulsatility may be due to increased forward wave intensity and not pressure from wave reflections.

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“…As it is done on body surface with a tape measure, obese people may have artificially longer distance. Although other methods for distance measurement were suggested, 11 the measurement on body surface is recommended by the expert documents. 7,10 The PWV measurement was also shown to have good reproducibility.…”

Section: Examinations and Materialsmentioning

confidence: 99%

The association between low 25-hydroxyvitamin D and increased aortic stiffness

et al. 2011

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There is accumulating evidence that vitamin D exerts important pathophysiological effects on cardiovascular system. Low vitamin D was associated with increased cardiovascular risk in several reports. We studied the association between vitamin D and arterial stiffness in a random sample of 560 subjects selected from general population. Arterial stiffness was measured as aortic pulse-wave velocity (PWV) using Sphygmocor device. Serum 25-hydroxyvitamin D (25(OH)D) was measured using commercial kits. We found a clear negative trend in aortic PWV among 25(OH)D quartiles. Subjects in the bottom 25(OH)D quartile (o20 ng ml À1 ) showed the highest aortic PWV (9.04 m s À1 ), compared with 2nd-4th quartile (8.07 m s À1 , 7.93 m s À1 and 7.70 m s À1 , respectively; P for trend o0.0001). The association between 25(OH)D and aortic PWV remained significant after adjustment for age, gender and other potential confounders; subjects in the first 25(OH)D quartile had adjusted odds ratio 2.04 (1.26-3.30) for having aortic PWV X9 m s À1 (top quartile) in multiple regression. In conclusion, we found a clear significant and independent negative association between 25(OH)D and aortic PWV. Subjects with lowest vitamin D status showed the highest arterial stiffness.

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The assessment of carotid–femoral distance for aortic pulse wave velocity: Should it be estimated from body height?☆

Cited by 10 publications

References 8 publications

Development and Validation of a Path Length Calculation for Carotid–Femoral Pulse Wave Velocity Measurement

Development and Validation of a Path Length Calculation for Carotid–Femoral Pulse Wave Velocity Measurement

Effect of acute resistance exercise on carotid artery stiffness and cerebral blood flow pulsatility

The association between low 25-hydroxyvitamin D and increased aortic stiffness

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