The anatomy of the spino‐cerebellar fibers in man. II. The distribution of the fibers in the cerebellum

Smith, Marion C.

doi:10.1002/cne.901170305

Cited by 13 publications

(1 citation statement)

References 30 publications

(10 reference statements)

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“…The 2 cases of iBL presented here were confirmed to have lesions in the lower lateral medulla by MRI. Patients with BL have been reported to have lesions in the dorsal spinocerebellar tract (DSCT), [3] , [4] the descending lateral vestibulospinal tract (LVST), the vestibulo-thalamic pathway (ascending graviceptive pathway), [5] the dentatorubrothalamic pathway, or the thalamocortical fascicle [6] . The DSCT and the LVST may be impaired by infarction in the medulla oblongata.…”

Section: Discussionmentioning

confidence: 99%

Isolated body lateropulsion caused by lower lateral medullary infarction

et al. 2017

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Section: Discussionmentioning

confidence: 99%

Isolated body lateropulsion caused by lower lateral medullary infarction

et al. 2017

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Spinal Cord and Brain Stem Pathways for Afferents from Joints

Gardner

1967

Ciba Foundation Symposium ‐ Myotatic, Kinesthetic and Vestibular Mechanisms

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Histogenesis of cortical layers in human cerebellum, particularly the lamina dissecans

Rakić

Sidman

1970

J of Comparative Neurology

374

234

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Patterns of lamination during development of the fetal human cerebellar cortex were analyzed in Nissl-and H & E-stained serial sections, rapid Golgi preparations, reduced silver impregnations, electron micrographs, and autoradiograrns. The layering pattern changed dramatically with time, as analyzed in detail for the culmen, the earliest region to differentiate. Up to about 10 weeks of gestation, cells proliferated only at or near the ventricular surface and migrated radially outward to occupy the full thickness of the cerebellar primordium except for an outermost cell-sparse marginal layer (2-layer stage). The external granular layer first appeared at 10-11 weeks while another group of cells became concentrated beneath the marginal layer (3-layer stage). At 20-21 weeks the lamina dissecans first became evident as a relatively acellular band in the midst of the zone of compact cells below the marginal (now molecular) layer, and for the next ten weeks the cerebellar cortex displayed this 5-layered form. At about 32 weeks the lamina dissecans disappeared (4-layer stage) and postnatally the external granular layer in turn disappeared as the last of its cells migrated inward (adult 3-layer configuration).The Purkinje cell population was established by 13 weeks, though the cerebellum

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The anatomy of the spino‐cerebellar fibers in man. II. The distribution of the fibers in the cerebellum

Cited by 13 publications

References 30 publications

Isolated body lateropulsion caused by lower lateral medullary infarction

Isolated body lateropulsion caused by lower lateral medullary infarction

Spinal Cord and Brain Stem Pathways for Afferents from Joints

Histogenesis of cortical layers in human cerebellum, particularly the lamina dissecans

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