Tachycardia‐Induced Cardiomyopathy: A Review of Literature

Khasnis, Atul; Jongnarangsin, Krit; Abela, George S.; Veerareddy, Srikar; Reddy, Vivek Y.; Thakur, Ranjan K.

doi:10.1111/j.1540-8159.2005.00143.x

Cited by 100 publications

(91 citation statements)

References 93 publications

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“…Należy odnotować, że objawy mogą być związane z dysfunkcją LV, ponieważ idiopatyczny VT może być przyczyną kardiomiopatii tachyarytmicznej [555]. U takich pacjentów powinno się rozważyć leki hamujące kanały sodowe (klasa IC) lub ablację przezcewnikową.…”

Section: Częstoskurcze Komoroweunclassified

2015 ESC Guidelines for the management of patients with ventricular arrhythmias and the prevention of sudden cardiac death

Priori

Blomström‐Lundqvist²,

Mazzanti³

et al. 2015

Kardiol Pol

233

337

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Section: Częstoskurcze Komoroweunclassified

2015 ESC Guidelines for the management of patients with ventricular arrhythmias and the prevention of sudden cardiac death

Priori

Blomström‐Lundqvist²,

Mazzanti³

et al. 2015

Kardiol Pol

233

337

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“…1 Tachycardia-induced cardiomyopathy can be defined as a condition of atrial or ventricular systolic and diastolic dysfunctions induced by increased atrial or ventricular rates, in the absence of prior history of structural heart disease. 2 Tachycardia-induced cardiomyopathy is highly dependent on the heart rate, and that higher heart rates induce TIC earlier. 3 -5 Adequate control of heart rate 6,7 or restoration of sinus rhythm 8,9 is necessary for improving cardiac function and symptoms.…”

Section: Introductionmentioning

confidence: 99%

Diagnostic Approach and Treatment Strategy in Tachycardia‐induced Cardiomyopathy

Jeong

Choi

Song

et al. 2008

Clinical Cardiology

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Background: Due to the absence of differential guidelines for heart failure with tachyarrhythmia, it is difficult to diagnose tachycardia-induced cardiomyopathy (TIC) at the initial visit. Furthermore, clinical outcomes of rate versus rhythm control in TIC are unclear. Hypothesis: Because the etiology of TIC is different from dynamic cardiomyoplasty (DCMP), differential parameters may be present. Methods: We assessed 21 patients with TIC (15 men; mean age, 50 ± 14 years) and 21 control patients with idiopathic DCMP. We assessed clinical courses, echocardiographic parameters, as well as outcomes by treatment.Results: In the TIC group, the related tachyarrhythmias were atrial fibrillation (n = 12), atrial flutter (n = 5), atrial tachycardia (n = 3) and paroxysmal supraventricular tachycardia (n = 1). After treatment, all patients became asymptomatic and the ejection fraction (EF) improvement ( EF 15%) was observed in all patients (left ventricular ejection fraction [LVEF], 30 ± 11% initial versus 58 ± 6% last ). In the idiopathic DCMP group, no patient showed EF improvement (EF increase 5%), and 4 patients (19%) underwent heart transplantation. Left ventricle (LV) mass indices, volumes adjusted by BSA, and dimensions were smaller in the TIC group than in the idiopathic DCMP group. Of those, LV end-diastolic dimension was the only independent predictor of TIC in multiple regression analysis (odds ratio [OR] 0.742 per 1 mm, 95% confidence ratio [CI] 0.618 to 0.891, p = 0.001). The Association of University Cardiologists (AUC) was 0.908 on receiver-operating characteristic (ROC) curve analysis and LV end-diastolic dimension 61 mm could predict TIC with a sensitivity of 100% and a specificity of 71.4%. After restoration of sinus rhythm (n = 8), one experienced recurrent TIC after discontinuation of amiodarone. After control of heart rate (n = 13), one experienced recurrent TIC due to poor control of heart rate (log-rank test, p = 0.808). There were no differences in the echocardiographic parameters between the 2 groups before and after treatment except for the larger initial LV volumes in the rhythm control. Conclusions: In patients presented as heart failure with tachyarrhythmia, initial echocardiographic parameters, especially LV end-diastolic dimension, help to differentiate TIC from idiopathic DCMP. Rate control was as effective as rhythm control for EF improvement and prognosis.

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“…This atrial kick often is lost because of the rapid atrial contraction that occurs when may develop as a result of disturbances in the structure and function of the left ventricle. 5 The resulting heart failure (HF) may be reversible once the ventricular rate is controlled; however, there is also a possibility that this myocardial damage may be irreversible. …”

Section: Hemodynamic Consequencesmentioning

confidence: 99%