T Waves in Hyperpotassemia

Braun, H; Surawicz, Borys; Bellet, Samuel

doi:10.1097/00000441-195508000-00005

Cited by 36 publications

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“…Clinically, electrocardiographic changes associated with hyperkalaemia have been most extensively studied in man. The first diagnostic signs are peaking of the T wave (Tarail 1948a;Reynolds, Martin and Homann 1951 ;Levine, Vazifelar, Lown and Merrill 1952;Braun, Surawicz and Bellet 1955) which may be associated with widening of the QRS complex (Tarail 1948b ;Braun et all955 ;Hendon et a1 1955). Broadening and flattening of the P wave also occurs (Tarail 1948b;Pick 1966;Wood 1968).…”

Section: Effect Of Potassium Chloride In Fusion On the Eccmentioning

confidence: 99%

“…The differences between experimental results and clinical observations can be explained by the interactions of other factors with hyperkalaemia in the clinical situation. In man, clinical hyperkalaemia is generally associated with renal insufficiency and, usually, uraemia (Fisch et all966) and precise clinical correlations between hyperkalaemia and the ECG do not always exist (Tarail 1948a;Braun et a/ 1955;Dreifus and Pick 1956).…”

Section: Effect Of Potassium Chloride In Fusion On the Eccmentioning

confidence: 99%

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Relationship between potassium administration, hyperkalaemia and the electrocardiogram: An experimental study

Epstein

1984

Equine Veterinary Journal

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Summary Hyperkalaemia affected the equine myocardium. The minimum plasma potassium concentration required to induce electrocardiographic changes was 6.2 mmol/litre and severe cardiotoxic effects were observed at levels of 8.0 to 10.1 mmol/litre in this experimental situation. The most consistent sign of hyperkalaemia was broadening and flattening of the P wave, which was generally associated with a change in T waves in the chest lead from negative to positive. The more pronounced the hyperkalaemia, the less pronounced the P wave and the more peaked positive the T wave. Severe hyperkalaemia was associated with various arrhythmias invariably associated with the presence of broad flat or absent P waves and upright T waves in chest leads. Caution against extrapolation from the experimental to the clinical situation must be exercised because of many complex interacting factors. Hyperkalaemia is difficult to induce experimentally and, unless associated with disease, is unlikely to occur in the horse.

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