Symptomatic Rathke's cleft cysts

Rout, D; Das, Liza; Rao, V. R. K.; Radhakrishnan, Venkatraman

doi:10.1016/0090-3019(83)90209-4

Cited by 42 publications

(18 citation statements)

References 22 publications

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“…Most often these cysts occured in the posterior fossa 1, 7, 8, 12, 16-19, 21, 23, 28, 29, 32, 33, 35, 37-39, 41, 44, 50, 53-56, 60, 62 Palma et al have reported the only case of suprasellar NC 44. The other suprasellar cysts reported in the literature, with the same histological findings as reported below and that of Palma et al, have always been called "ectopic suprasellar Rathke's cleft cysts TM, 5,13,26,27,47,57,58,61 …”

Section: Introductionsupporting

confidence: 52%

Do the suprasellar neurenteric cyst, the Rathke cleft cyst and the colloid cyst constitute a same entity?

et al. 1995

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Two cases of entirely suprasellar cysts are reported. Total surgical removal was performed in both cases. Pathological and immunohistochemical profiles were consistent with neurenteric cysts, Rathke's cleft cysts or colloid cysts and was also in keeping with an endodermal origin. It is now admitted that these three kinds of cysts share similar histological and immunohistological features. We propose an hypothesis of common embryological origin from endodermal remnants.

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Section: Introductionsupporting

confidence: 52%

Do the suprasellar neurenteric cyst, the Rathke cleft cyst and the colloid cyst constitute a same entity?

et al. 1995

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“…Thus, all structures giving rise to the adenohypophysis and neurohypophysis are of ectodermal origin. rences necessitated reoperation from 4 months to 26 years after initial resection.16.2(I, 23,27,[33][34][35]52 Given the infrequency with which these lesions appear in the literature, this number of recurrences is surprising. Voelker, et al, 4~ estimated a 5% to 10% recurrence rate for Rathke's cleft cyst in their review of the literature, but did not state how this figure was calculated.…”

Section: Correlation Of Histology and Clinical Behaviormentioning

confidence: 88%

Epithelial cystic lesions of the sellar and parasellar region: a continuum of ectodermal derivatives?

Harrison

Morgello²,

Post³

1994

Journal of Neurosurgery

158

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Cystic epithelial lesions of the sellar and parasellar region are classified on the basis of histology and location into Rathke's cleft cysts, epithelial cysts, epidermoid cysts, dermoid cysts, and craniopharyngiomas. A retrospective review of the clinical presentation, radiological findings, and histology was performed on 19 such lesions, and a survey of the literature pertinent to the classification, clinical presentation, and embryology of these lesions was conducted. Presentation was nonspecific and not predictive of histology. Imaging studies were generally useful in distinguishing these tumors, with the exception of Rathke's cleft cysts, suprasellar epidermoid cysts, and craniopharyngiomas, which frequently could not be differentiated. On microscopic examination, most lesions fit into distinct categories; however, overlap was common among all and some could not be definitively categorized by histological criteria. Evidence supportive of an ectodermal ancestry for sellar and parasellar epithelial-lined cystic lesions is presented. Based on the current findings and a review of the literature, it is suggested that these lesions represent a continuum of ectodermally derived cystic epithelial lesions.

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“…The histological distinction between craniopharyngioma and Rathke's cleft cyst is not made easily in our patient; however, it is important to distinguish them because of the contrast in their clinical behavior [11,12]. Craniopharyngiomas are aggressive tumors with a tendency for regrowth after incomplete removal.…”

Section: Discussionmentioning

confidence: 99%

A Boy with Rathke's Cleft Cyst

Okada¹,

Hashimoto²,

Koga³

et al. 1993

Clin Pediatr Endocrinol

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Abstract. We report a 13-year-old boy with Rathke's cleft cyst who had diabetes insipidus (DI), visual disturbance, and GH deficiency. He was admitted to our hospital in 1988 because of polyuria and visual disturbance. He had homonymous hemianopsia and DI; however, the brain computed tomography (CT) was normal. He received 1-deamino-8-D-arginine vasopressin (DDAVP) therapy. In 1989, he was found to have GH deficiency and GH treatment was started. In 1991, a 1.2 cm sized mass was found in the sella area by magnetic resonance imaging (MRI) and removed by microscopic transsphenoidal surgery. Histological examination revealed a Rathke's cleft cyst with craniopharyngioma.In spite of visual disturbance at the first visit, no mass had been found in the brain by CT or MRI during the first two years of GH treatment. However, the mixed form of Rathke's cleft cyst and craniopharyngioma had grown rapidly and was subsequently detected by MRI. He has to be followed up very carefully by MRI for a long period.

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Symptomatic Rathke's cleft cysts

Cited by 42 publications

References 22 publications

Do the suprasellar neurenteric cyst, the Rathke cleft cyst and the colloid cyst constitute a same entity?

Do the suprasellar neurenteric cyst, the Rathke cleft cyst and the colloid cyst constitute a same entity?

Epithelial cystic lesions of the sellar and parasellar region: a continuum of ectodermal derivatives?

A Boy with Rathke's Cleft Cyst

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