Stimulator selection in SSVEP-based BCI

Wu, Zhenghua; Lai, Yongxiu; Yang, Xiaobo; Wu, Dan; Yao, Dezhong

doi:10.1016/j.medengphy.2008.01.004

Cited by 159 publications

(86 citation statements)

References 16 publications

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“…An SSVEP response is possible with a large number of frequencies, ranging from 1Hz to 100Hz, with resonance peaks at 10, 20, 40 and 80Hz [4]. The spectrum differences of three kinds of flickers and the differences in SSVEPs evoked by three different stimulators, the light-emitting diode, the cathode ray tube of a desktop monitor and the liquid crystal display of a laptop screen have been investigated in [11]. The amplitude of SSVEP response evoked by LEDs is significantly larger than that evoked by stimuli on the monitors.…”

Section: Ssvep Stimuli On Lcd Screenmentioning

confidence: 99%

Impact of Frequency Selection on LCD Screens for SSVEP Based Brain-Computer Interfaces

Volosyak

Cecotti

Gräser

2009

Lecture Notes in Computer Science

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Abstract. In this work, the high impact of appropriate selection of visual stimuli on liquid crystal displays (LCDs) used for the brain-computer interfaces (BCIs) based on the Steady-State Visual Evoked Potentials (SSVEPs) has been confirmed. The number of suitable frequencies on the standard LCD monitor is limited due to the vertical refresh rate of 60Hz and the number of simultaneously used stimuli. Two sets of frequencies have been compared among each other during the on-line spelling task with the Bremen-BCI system in the study with 10 healthy subjects. This work is meaningful for the practical design of LCD based BCIs. In this study, appropriate selection of visual stimuli results in a 40% change in the BCI literacy under otherwise equal conditions.

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Section: Ssvep Stimuli On Lcd Screenmentioning

confidence: 99%

Impact of Frequency Selection on LCD Screens for SSVEP Based Brain-Computer Interfaces

Volosyak

Cecotti

Gräser

2009

Lecture Notes in Computer Science

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“…Because the phase and the amplitude of the SSVEP response depend on the frequency, structure, and intensity of the repetitive visual clues [37], to choose the type of visual clues is an extremely important problem. This problem needs to be determined first in SSVEP-based BCI.…”

Section: A Visual Stimulusmentioning

confidence: 99%

SSVEP based brain-computer interface controlled functional electrical stimulation system for upper extremity rehabilitation

Chu

Zhao

Han

et al. 2014

2014 IEEE International Conference on Robotics and Biomimetics (ROBIO 2014)

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“…A more recent study [23] showed that PERGs are virtually unaffected in MSA, whereas in early PD they are clearly impaired, suggesting different pathogenic retinal mechanisms and a useful simple tool for distinguishing MSA from PD. The strongest objection against the use of VEPs for the assessment of synucleinopathies derived from the technical constraints of VEP recordings: VEPs are altered by abnormalities of optic nerve and visual pathways, VEPs recordings require the adequate collaboration of patients who must focus attention on stimuli [24][25][26], VEP variable (amplitude, latency) are dependent on laboratories settings and must be adjusted according to each laboratory statistics of distribution. The characteristic of VEP cannot be simply shared by different laboratories and differences in equipments might sustain variability which are far wider than variability observed in patient and control populations.…”

Section: Visual Evoked Potentials (Veps)mentioning

confidence: 99%

Is There a Place for Clinical Neurophysiology Assessments in Synucleinopathies?

Onofrj¹,

Bonanni²,

Thomas³

et al. 2011

Neuroimaging for Clinicians - Combining Research and Practice

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IntroductionParkinson Disease (PD), Dementia with Lewy Bodies (DLB) and Multiple System Atrophy (MSA) are characterized by deposition of Lewy Bodies (LB), consisting of eosinophilic intracellular (intraneural in PD and DLB and intraglial in MSA) inclusions of -synuclein and allowing the three disorders to be categorized as synucleinopathies. The identification in the last two decades of specific clinical features of synucleinopathies has been a major breakthrough in Neurology, as it prompted a reconsideration of diagnostic and therapeutic approaches to dementia and parkinsonism. The recognition of synuclein and ubiquitin markers in dementia resulted in the reclassification of patients previously considered as affected by Alzheimer Disease (AD) and Vascular Dementia (VaD), and there is now agreement that DLB is the second most common cause of dementia in elderly population: its prevalence is reported to be in the 25-43% range in different studies. DLB is clinically characterized by the presence of prominently dysexecutive dementia (frontal lobe or subcortical dementia according to earlier classification [1]), cognitive fluctuations, consisting of remitting-relapsing episodes of blunted conscience reaching levels of stupor, by the occurrence of visual hallucinations and delusions, by parkinsonian motor signs and hypersensitivity to neuroleptic treatment, ranging from worsening of parkinsonism to the possible lethal neuroleptic-malignant syndromes [2]. Yet, variances of presentation and overlapping symptoms with AD and Fronto-Temporal lobe Degeneration (FTD), could be misleading in the process of addressing a clinical diagnosis with consequent therapeutic risks(e.g. introducing neuroleptic treatments in patient with DLB), or economic costs (e.g. addressing patients with DLB to treatment protocols dedicated to AD patients, based on vaccines or antibodies against amyloid proteins, a neuropathological feature of AD). It is evident the stringent need for improvement of reliable diagnostic tools (u.e. biomarkers) to differentiate the diseases associated with dementia. In 2010 the National Institute of Health, NIH, held a symposium focused on the development of possible biomarkers for DLB. Among the different suggested biomarkers, neurophysiological assessments were reconsidered, as a large amount of neurophysiological studies had been devoted to AD and PD, whose characteristics are shared by DLB. www.intechopen.com Neuroimaging for Clinicians -Combining Research and Practice 300We contributed to several studies on this argument along the years, and in the present chapter we discuss the role of clinical neurophysiological studies in DLB in comparison with other disorders. The essential background of our original studies laid in the fact that most historical studies were performed when the DLB clinical entity was unknown and therefore some of the past results were marred by absent recognition of this clinical entity. Synucleinopathies: Dementia with lewy bodiesSynucleinopathies comprise a diverse groups of neurodegenerati...

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Stimulator selection in SSVEP-based BCI

Cited by 159 publications

References 16 publications

Impact of Frequency Selection on LCD Screens for SSVEP Based Brain-Computer Interfaces

Impact of Frequency Selection on LCD Screens for SSVEP Based Brain-Computer Interfaces

SSVEP based brain-computer interface controlled functional electrical stimulation system for upper extremity rehabilitation

Is There a Place for Clinical Neurophysiology Assessments in Synucleinopathies?

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