St�rungen des Lichtsinns und Farbensinns bei Chorioretinitis centralis serosa

Jäeger, Wolfgang; Nover, A

doi:10.1007/bf00683815

Cited by 31 publications

(8 citation statements)

References 3 publications

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“…Central serous chorio-retinopathy characteristically shows protanomalous Rayleigh matches, which becomes progressively more severe with deterioration of the condition (Kitahara, 1936;Jaeger & Noyer, 1951 The apparatus, matching procedures, and calibrations are described in detail by Alpern et al (1976). Colour matches were made between a 10 central disk and a contiguous annular surround with 30 outside diameter.…”

Section: Nagel Anomaloscopementioning

confidence: 99%

Classical tritanopia

Alpern

Kono

Krantz

1983

The Journal of Physiology

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6. Colour matches made by the trichromatic eye do not match when viewed by the tritanopic eye, almost certainly because the ocular media of the two eyes have wave-length-dependent differences in absorption. For the largest difference (430 nm) the trichromatic eye transmits about 2-2 times more light than its fellow. When allowance is made for these differences, the field sensitivities of n4 and H5 of the two eyes do not differ. The field sensitivities of n4 and H5 of the normal eye, on the other hand, differ significantly from those of the average spectra obtained on four normal trichromats by Stiles, in a way that cannot be attributed to differences in transmittance of ocular media.7. It is concluded that classical (or acquired) tritanopia is not distinguishable in its

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Section: Nagel Anomaloscopementioning

confidence: 99%

Classical tritanopia

Alpern

Kono

Krantz

1983

The Journal of Physiology

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“…In CSC, diminished red sensitivity is an early event [4,22]. Pseudoprotanomaly may be present even with normal visual acuity [6] ( Table 4).…”

Section: Discussionmentioning

confidence: 99%

Anomaloscope examination in macular gliosis, macular holes and central serous choroidopathy

Tilanus

Pinckers

Aandekerk

1998

Graefe's Archive for Clinical and Experimental Ophthalmology

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“…The anterior displacement of the retinal plane may account for hypermetropization, and irregularities in the shape of the retinal plane may account for metamorphopsia. The cause of micropsia is unknown, but it has been suggested that this may be due to the hypermetropization in CSC (Jaeger & Nover 1951), or be a result of an increased distance between photoreceptors in the detached retinal area (Frisen & Frisen 1979). The present findings confirm previous studies that the most prominent colour vision abnormality in CSC is pseudo-protanomaly (Jaeger & Nover 1951;Legras & Coscas 1971).…”

Section: Discussionmentioning

confidence: 99%

“…central serous choroidopathy (CSC). CSC is characterized by a central serous detachment accompanied by a pigment epithelial defect or detachment as seen by fluorescein angiography and OCT (Hee et al 1995b), and the disease is often accompanied by dyschromatopsia that can be classified as pseudo-protanomaly (Jaeger & Nover 1951;Legras & Coscas 1971;Tilanus et al 1998). The pathophysiolog-ical mechanisms underlying this change in colour perception in CSC is unknown, but may be due to either an optical or a neuronal disturbance in the diseased retina.…”

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confidence: 99%

Quantitative anomaloscopy and optical coherence tomography scanning in central serous chorioretinopathy

Bek

Kandi

2000

Acta Ophthalmologica Scandinavica

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ABSTRACT.Background: Dyschromatopsia is a prominent sign in a variety of central retinal diseases, such as central serous chorioretinopathy (CSC). The changes in colour vision may be due to either optical or neuronal factors in the diseased retina. The relative contribution from the two causes is unknown, but may be elucidated by obtaining knowledge of the anatomical derangement in the diseased retina in CSC. Methods: Twenty-six normal persons had their colour vision tested using the Tomey anomaloscope. The calculation of setting range (SR) and central mean point (CMP) for Rayleigh match and Moreland match was optimized, and normal ranges for these values were defined. Subsequently 24 patients with CSC were examined by anomaloscopy and optical coherence tomography scanning, and the measures of colour vision were related to the anatomical changes observed on the scans. Results: The algorithm for calculating SR and CMP which is integrated into the Tomey anomaloscope could be considerably improved to increase sensitivity and reproducibility of these measures. Fifteen patients had abnormal colour vision. Nine patients had pseudo-protanomaly, seven patients had pseudo-tritanomaly, and three patients had abnormalities in both matches. There was no relation between these colour vision abnormalities and the anatomical derangement as seen by OCT in the diseased central retina. Conclusion:The findings argue against the notion that the density of retinal cell nuclei, the orientation of photoreceptors, or the size of the central serous detachment are related to the colour vision abnormalities in CSC. The question of whether these abnormalities are due to optical or neuronal factors remains open.

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St�rungen des Lichtsinns und Farbensinns bei Chorioretinitis centralis serosa

Cited by 31 publications

References 3 publications

Classical tritanopia

Classical tritanopia

Anomaloscope examination in macular gliosis, macular holes and central serous choroidopathy

Quantitative anomaloscopy and optical coherence tomography scanning in central serous chorioretinopathy

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