Dan Chen, Zhi-qi Gao, Ying-ying Wang, Bin-bin Wan, Gang Liu, Jun-liang Chen, Ya-xian Wu, Qin Zhou, Shan-yu Jiang, Ren-qiang Yu, Qing-feng Pang
Alveolar arrest and the impaired angiogenesis caused by chronic inflammation and oxidative stress are two main factors in bronchopulmonary dysplasia (BPD). Short-chain fatty acids (SCFAs), especially propionate, possess anti-oxidant and anti-inflammatory effects. The present study was designed to examine the roles of sodium propionate (SP) on lipopolysaccharide (LPS)-challenged BPD and its potential mechanisms.
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