Single‐cause Attribution

Leviton, Alan

doi:10.1111/j.1469-8749.1987.tb08829.x

Cited by 27 publications

(6 citation statements)

References 22 publications

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“…PE is a syndrome [15], as are all pregnancy complications and neurodevelopmental disorders [16]. This helps to explain why the risk of CP and other indicators of perinatal brain damage or dysfunction associated with PE differs greatly depending on the length of gestation at delivery, its fetoplacental correlates, related postnatal systemic inflammation-related phenomena and the background incidence of other disorders.…”

Section: Resultsmentioning

confidence: 99%

The So-Called “Protective Effect” of Preeclampsia on Perinatal Brain Damage

Korzeniewski¹

2018

Neonatology

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Section: Resultsmentioning

confidence: 99%

The So-Called “Protective Effect” of Preeclampsia on Perinatal Brain Damage

Korzeniewski¹

2018

Neonatology

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“…Fourth, we allow for multiple causes to be involved in disease etiology instead of looking for single culprits. 7 A more complex example is the causation of cerebral palsy (CP). The most recent definition holds that CP encompasses 'a group of disorders of the development of movement and posture, causing activity limitation, that are attributed to nonprogressive disturbances that occurred in the developing fetal or infant brain'.…”

Section: Reviewmentioning

confidence: 99%

Evidence-based child neurology

Dammann¹

2006

Dev Med Child Neurol

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This paper discusses some of the issues that, once resolved, may help child neurologists and pediatric neuroepidemiologists in their mutual understanding and collaborative efforts. Emphasis is placed on the all-too-frequent assumption that evidence-based medicine only covers the application of results from randomized double-blind trials in practice. A considerable proportion of child neurology concerns clinical diagnosis and etiological inference. Therefore, evidence-based child neurology might benefit not only from the therapeutic, but also from the diagnostic and etiological components of epidemiology.

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“…Der Umkehrschluss hingegen, dass perinatale Hirnschäden vornehmlich hypoxischischämischer Ursache sind, ist nicht zulässig. [14] und die Tatsache, dass nur ein sehr geringer Teil der Frühgeborenen mit einem vermeintlich hypoxisch-ischämischen Ereignis in der Krankengeschichte eine Hirnschädigung erleiden, sprechen eher dafür, dass die ¾tiologie perinataler Hirnschäden komplex ist [15] und daher von einer Einzelursachenzuweisung Abstand genommen werden sollte [16].…”

Section: Hypoxie-ischämie Vs Inflammationunclassified

Intrauterine Infektion und das Gehirn Frühgeborener - Dimensionen der Ätiologieforschung

Dammann

2006

Z Geburtshilfe Neonatol

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Perinatal brain damage has a diverse and complex aetiology. Over the past decades, much progress has been made in this research field. In this article, I offer a discussion of seven dimensions of aetiological perinatal brain damage research: (1) hypoxia-ischaemia vs. inflammation; (2) "classic" vs. "remote" intrauterine infection; (3) focal vs. diffuse white matter damage; (4) maternal vs. foetal inflammatory response; (5) clinical vs. experimental data; (6) bacterial vs. viral infection; and (7) preterm vs. term delivery. Despite these complexities, it is hoped that obstetricians, neonatologists, and neuropaediatricians will agree on a perinatal neuroprotective strategy in the near future.

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Single‐cause Attribution

Cited by 27 publications

References 22 publications

The So-Called “Protective Effect” of Preeclampsia on Perinatal Brain Damage

The So-Called “Protective Effect” of Preeclampsia on Perinatal Brain Damage

Evidence-based child neurology

Intrauterine Infektion und das Gehirn Frühgeborener - Dimensionen der Ätiologieforschung

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