2008
DOI: 10.1590/s0066-782x2008000500015
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Abstract: Eisenmenger's syndrome consists of pulmonary hypertension with a reversed or bidirectional shunt at the atrioventricular, or aortopulmonary level. Eisenmerger's syndrome in pregnancy is usually associated with high mortality rates (nearly 30-50%). Unfortunately, pulmonary hypertension is aggravated during pregnancy and often leads to an unfavorable outcome. Here, we report a successful pregnancy in a woman with Eisenmenger syndrome.

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Cited by 10 publications
(5 citation statements)
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“…Once the patients develops Eisenmenger syndrome, the machinery murmur might be unaudible and the associated patent ductus arteriosus might be misdiagnosed [10] . Patients may have a low oxygen saturation [11] and polycythemia [12] . Severe complications, such as heart failure, endocarditis and thromboembolic accidents, may develop in the condition of pregnancy.…”
Section: Clinical Manifestationmentioning
confidence: 99%
“…Once the patients develops Eisenmenger syndrome, the machinery murmur might be unaudible and the associated patent ductus arteriosus might be misdiagnosed [10] . Patients may have a low oxygen saturation [11] and polycythemia [12] . Severe complications, such as heart failure, endocarditis and thromboembolic accidents, may develop in the condition of pregnancy.…”
Section: Clinical Manifestationmentioning
confidence: 99%
“…Pulmonary hypertension in pregnancy is associated with high mortality (30%-50%);[123] hence, pregnancy is contraindicated. Whatever anesthetic technique is chosen, principle remains the same.…”
Section: Discussionmentioning
confidence: 99%
“…However, in patients with ES, an irreversibly increased pulmonary vascular resistance restricts blood flow to the lungs. Therefore, an increased plasma volume adds burden to the already compromised right ventricle [ 3 , 4 ]. Moreover, systemic vasodilation with pre-existing pulmonary hypertension increases the right-to-left shunt, which worsens the hypoxia [ 3 , 4 ].…”
mentioning
confidence: 99%
“…Therefore, an increased plasma volume adds burden to the already compromised right ventricle [ 3 , 4 ]. Moreover, systemic vasodilation with pre-existing pulmonary hypertension increases the right-to-left shunt, which worsens the hypoxia [ 3 , 4 ]. These series of changes occur in a vicious cycle of increases in the pulmonary hypertension, right ventricular strain, and right-to-left shunt [ 3 ].…”
mentioning
confidence: 99%
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