Serum Hepatocyte Growth Factor Predicts Ventricular Remodeling Following Myocardial Infarction.

Hepatocyte Growth Factor and Cardiovascular Thrombosis in Patients Admitted to the Intensive Care Unit

“…Other investigators found that there is an increase in serum HGF in patients with AMI and unstable angina. 26,27,29,35,36 Our results are similar to those reports. Soeki et al suggested that the serum HGF concentration peaked at 7 days after the onset of AMI, and it was related to left ventricular remodeling.…”

“…Soeki et al suggested that the serum HGF concentration peaked at 7 days after the onset of AMI, and it was related to left ventricular remodeling. 35 Shimada et al reported that the serum HGF concentration on the 7th day of AMI had a relationship with C-reactive protein, and that HGF was involved in the inflammatory reaction. 36 However, in the present study, the serum HGF concentration was measured only on arrival at the hospital, because Matsumori et al found that the serum HGF concentration increased after heparin administration.…”

Diagnostic validity of hepatocyte growth factor as marker for rejection in the follow-up of patients after heart transplantation

“…14 Results by Soeki et al confirm this hypothesis, because in patients with acute myocardial infarction, there was an increased serum HGF concentration and no difference between HGF serum levels measured in the coronary sinus and in the periphery. 11 Therefore, these results suggest that the increased serum HGF concentration may result from its release to the bloodstream not only from the cells infiltrated heart, but also from other organs. Furthermore, we found any correlation between serum HGF and NP concentrations.…”

Mechanism of Myocardial Microvessel Formation in Cyanotic Congenital Heart Disease

“…[2][3][4][11][12][13][14][15] In clinical practice, abnormal blood vessels are a cause of significant morbidity during or after the surgical management of congenital heart disease. Abnormal collateral vessel development in children with C-CHD may take several forms, including systemic venous collaterals, which may be extensive, pulmonary arteriovenous malformations, or aortopulmonary collateral arteries.…”