Seminal megavesicles with adult polycystic kidney disease

Hendry, W. F.; Rickards, D.; Pryor, John P.; Baker, L. R. I.

doi:10.1093/humrep/13.6.1567

Cited by 42 publications

(29 citation statements)

References 14 publications

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“…Vasography shows mega-vesicles, with no visible obstruction. Hendry et al [45] recently showed that dilated SV in hypofertile men with polycystic kidney disease was not due to cysts, but to SV atonia responsible for a functional obstacle on the ejaculatory ducts (Fig. 5d).…”

Section: Ejaculatory Duct Obstructionmentioning

confidence: 93%

US findings in male hypofertility and impotence

et al. 2001

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IntroductionUltrasonography of the male genital tract is routinely performed to assess several diseases in man. A major field is prostate transrectal sonography combined with US-guided biopsies used to improve, in combination with PSA assay, early detection and staging of prostate cancer. Two other fields have gained clinical importance. The first is male hypofertility. Improvement of in vitro fertilization techniques has led to a huge increase in pretreatment investigation by imaging of hypofertile men, with emphasis on azoospermic patients, to try to find the cause of infertility, before contemplating invasive techniques for only diagnostic purposes, such as testicular biopsy or surgical deferentography. It also concerns non-azoospermic patients in whom the search for a deleterious factor is more and more routinely conducted to improve the quality of the semen. Scrotal and transrectal ultrasonography with high-frequency probes and use of color Doppler imaging have proved to be very reliable adjuncts to clinical examination to assess the presence of an obstruction (total or partial) of the seminal tract, to detect inflammatory changes in the deep genital tract and to detect and quantify a venous reflux in the spermatic veins. The second field is male sexual impotence. Initial enthusiasm concerning noninvasive investigation of impotent men by color Doppler sonography to detect a vascular abnormality (arterial or venous) of the penile vasculature has been tempered by the fact that surgical or interventional treatments used to treat penile artery stenoses or incompetence of the veno-occlusive system were found to give poor results, and now these treatments have been superseded by the advent of medical treatments based on intracavernosal injection of vasoactive drugs and more recently by the possibility of circumventing vasculogenic impotence by a peroral treatment, namely Viagra. US has thus become useful in only highly selected patients presenting with sexual impotence.

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Section: Ejaculatory Duct Obstructionmentioning

confidence: 93%

US findings in male hypofertility and impotence

et al. 2001

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“…Most authors reporting an association with ADPKD describe bilateral seminal vesicle cysts, although it is not certain whether these are acquired or congenital. Marked distension of the seminal vesicles (i.e., megavesicles) is a separate entity, described previously in a small number of studies in ADPKD patients [5][6][7]13]. The majority of previous reports of seminal vesicle dilatation have been in patients presenting with hematospermia but no known ADPKD [14,15].…”

Section: Discussionmentioning

confidence: 83%

“…The reported prevalence of seminal vesicle cysts in patients with ADPKD varies widely, from 6% by computed tomography (CT) [3] to 21%-60% by transrectal ultrasound [1,4]. Seminal vesicle dilatation is rarely reported in patients with ADPKD but has been found during infertility evaluations [5][6][7]. Mean seminal vesicle diameter has been reported to be greater in ADPKD patients than normal subjects by CT criteria [8].…”

Section: Introductionmentioning

confidence: 99%

Seminal megavesicle in autosomal dominant polycystic kidney disease

et al. 2015

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“…Hendry et al [ 28 ] described 6 patients with this fi nding and determined that the gross dilatation of the seminal vesicles seen on TRUS was not caused by obstruction but by atonicity. These ultrasonic appearances, when described previously, were incorrectly thought to be due to seminal vesicle cysts.…”

Section: Seminal Vesiclesmentioning

confidence: 91%