School mobility during childhood predicts psychotic symptoms in late adolescence

Winsper, Catherine; Wolke, Dieter; Bryson, Alex; Thompson, Andrew; Singh, Swaran P.

doi:10.1111/jcpp.12572

Cited by 18 publications

(12 citation statements)

References 57 publications

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“…A more positive atmosphere at home was also found to be protective in the poly-victimized group and among the general population, which is consistent with prior research that has highlighted the protective effects of family stability in the context of adversity, 62 and how more chaotic living situations can increase the risk of early psychotic symptoms 63 and adult psychosis. 64 Given that some types of victimization that children are exposed to may take place outside of the home, the home environment may provide children with a safe, nurturing environment that acts as a refuge, which, in turn, may lessen the harmful effects of their experiences on cognitive and emotional processes.…”

Section: Discussionsupporting

confidence: 88%

Protective Factors for Psychotic Symptoms Among Poly-victimized Children

Crush

Arseneault

Jaffee

et al. 2017

Schizophrenia Bulletin

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BackgroundExperiencing victimization in early life has been repeatedly shown to be associated with the emergence of psychotic symptoms in childhood. However, most victimized children do not develop psychotic symptoms and why this occurs is not fully understood. This study investigated which individual, family-level, and wider community characteristics were associated with an absence of psychotic symptoms among children at risk for psychosis by virtue of their exposure to multiple victimization experiences (poly-victimization).MethodsParticipants were from the Environmental Risk Longitudinal Twin Study, a nationally representative cohort of 2232 UK-born twins. Exposure to maltreatment, bullying and domestic violence prior to age 12 was determined from interviews with mothers, children, and observations by research workers at ages 5, 7, 10, and 12. Children were interviewed about psychotic symptoms at age 12. Protective factors were measured at ages 5, 7, 10, and 12.ResultsChildhood poly-victimization was associated with age-12 psychotic symptoms (OR = 4.61, 95% CI 2.82–7.52), but the majority of poly-victimized children did not report symptoms (80.7%). Having a relatively high IQ, more positive atmosphere at home, and higher levels of neighborhood social cohesion were found to be protective against childhood psychotic symptoms among poly-victimized children and also in the whole sample. However, “protected” poly-victimized children displayed elevated levels of other mental health problems compared to nonvictimized children.ConclusionsChildren’s characteristics, family context, and the wider community were all found to protect children from developing early psychotic symptoms, even when they were victimized multiple times. These findings indicate targets for multilevel preventive interventions.

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Section: Discussionsupporting

confidence: 88%

Protective Factors for Psychotic Symptoms Among Poly-victimized Children

Crush

Arseneault

Jaffee

et al. 2017

Schizophrenia Bulletin

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“…A direct significant association between bully victimisation and BPD symptoms was also observed. While bully victimisation has been linked to a multitude of negative mental health outcomes [50, 51], this study reveals a prospective link between bully victimisation and BPD specifically, adding to an emerging evidence base highlighting the importance of peer relationships in the development of BPD [23, 15]. …”

Section: Discussionmentioning

confidence: 86%

Aetiological pathways to Borderline Personality Disorder symptoms in early adolescence: childhood dysregulated behaviour, maladaptive parenting and bully victimisation

Winsper

Hall

Strauss

et al. 2017

bord personal disord emot dysregul

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BackgroundDevelopmental theories for the aetiology of Borderline Personality Disorder (BPD) suggest that both individual features (e.g., childhood dysregulated behaviour) and negative environmental experiences (e.g., maladaptive parenting, peer victimisation) may lead to the development of BPD symptoms during adolescence. Few prospective studies have examined potential aetiological pathways involving these two factors.MethodWe addressed this gap in the literature using data from the Avon Longitudinal Study of Parents and Children (ALSPAC). We assessed mother-reported childhood dysregulated behaviour at 4, 7 and 8 years using the Strengths and Difficulties Questionnaire (SDQ); maladaptive parenting (maternal hitting, punishment, and hostility) at 8 to 9 years; and bully victimisation (child and mother report) at 8, 9 and 10 years. BPD symptoms were assessed at 11 years using the UK Childhood Interview for DSM-IV BPD. Control variables included adolescent depression (assessed with the Short Moods and Feelings Questionnaire-SMFQ) and psychotic symptoms (assessed with the Psychosis-Like Symptoms Interview-PLIKS) at 11 to 14 years, and mother’s exposure to family adversity during pregnancy (assessed with the Family Adversity Scale-FAI).ResultsIn unadjusted logistic regression analyses, childhood dysregulated behaviour and all environmental risk factors (i.e., family adversity, maladaptive parenting, and bully victimisation) were significantly associated with BPD symptoms at 11 years. Within structural equation modelling controlling for all associations simultaneously, family adversity and male sex significantly predicted dysregulated behaviour across childhood, while bully victimisation significantly predicted BPD, depression, and psychotic symptoms. Children displaying dysregulated behaviour across childhood were significantly more likely to experience maladaptive parenting (β = 0.075, p < 0.001) and bully victimisation (β = 0.327, p < 0.001). Further, there was a significant indirect association between childhood dysregulated behaviour and BPD symptoms via an increased risk of bullying (β = 0.097, p < 0.001). While significant indirect associations between dysregulated behaviour, bully victimisation and depression (β = 0.063, p < 0.001) and psychotic (β = 0.074, p < 0.001) outcomes were also observed, the indirect association was significantly stronger for the BPD outcome (BPD – depression = 0.034, p < 0.01; BPD – psychotic symptoms = 0.023, p < 0.01).ConclusionsChildhood dysregulated behaviour is associated with BPD in early adolescence via an increased risk of bully victimisation. This suggests that childhood dysregulation may influence the risk of bully victimisation, which in turn influences the development of BPD. Effective interventions should target dysregulated behaviour early on to reduce exposure to environmental risks and the subsequent development of BPD.

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“…The pattern and comparative strength of associations were very similar in the weighted to unweighted analysis. We, therefore, report the unweighted analysis (Winsper, Wolke, Bryson, Thompson, & Singh, ).…”

Section: Methodsmentioning

confidence: 99%

Associations between infant and toddler regulatory problems, childhood co‐developing internalising and externalising trajectories, and adolescent depression, psychotic and borderline personality disorder symptoms

Winsper

Bilgin

Wolke

2019

Child Psychology Psychiatry

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Background Early regulatory problems (RPs) are associated with childhood internalising and externalising symptoms. Internalising and externalising symptoms, in turn, are associated with adolescent psychopathology (e.g. personality disorders, depression). We examined whether RPs are directly associated with adolescent psychopathology, or whether associations are indirect via childhood internalising and externalising symptoms. Methods We used data from the Avon Longitudinal Study of Parents and Children. Mothers reported on their child's RPs at 6, 15–18 and 24–30 months, and internalising and externalising symptoms at 4, 7, 8 and 9.5 years. Adolescent psychotic, depression and BPD symptoms were assessed at 11–12 years. Children were grouped by their patterns of co‐developing internalising and externalising symptoms using parallel process latent class growth analysis (PP‐LCGA). Path analysis was used to examine direct and indirect associations from RPs to the three adolescent outcomes. Results There were four groups of children with distinct patterns of co‐developing internalising and externalising (INT/EXT) symptoms. Most children (53%) demonstrated low–moderate and stable levels of INT/EXT symptoms. A small proportion (7.7%) evidenced moderate and increasing INT and high stable EXT symptoms: this pattern was strongly predictive of adolescent psychopathology (e.g. depression at 11 years: unadjusted odds ratio = 5.62; 95% confidence intervals = 3.82, 8.27). The other two groups were differentially associated with adolescent outcomes (i.e. moderate–high increasing INT/moderate decreasing EXT predicted mother‐reported depression at 12, while low stable INT/moderate–high stable EXT predicted child‐reported depression at 11). In path analysis, RPs at each time‐point were significantly indirectly associated with symptoms of BPD and child‐ and mother‐reported depression symptoms via the most severe class of INT/EXT symptoms. Conclusions Consistent with a cascade model of development, RPs are predictive of higher levels of co‐developing INT/EXT symptoms, which in turn increase risk of adolescent psychopathology. Clinicians should be aware of, and treat, early RPs to prevent chronic psychopathology.

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School mobility during childhood predicts psychotic symptoms in late adolescence

Cited by 18 publications

References 57 publications

Protective Factors for Psychotic Symptoms Among Poly-victimized Children

Protective Factors for Psychotic Symptoms Among Poly-victimized Children

Aetiological pathways to Borderline Personality Disorder symptoms in early adolescence: childhood dysregulated behaviour, maladaptive parenting and bully victimisation

Associations between infant and toddler regulatory problems, childhood co‐developing internalising and externalising trajectories, and adolescent depression, psychotic and borderline personality disorder symptoms

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