Role of Micronutrients in Heart Diseases

Abstract

Heart disease is a common occurrence in older patients in the civilized culture, and the rate is predicted to rise as the software advances. Patients with heart disease should be intended to eat a salt-free diet to lose adiposity. Diet is also critical for heart disease patients; those with nutrition deficits have a low deep prognosis. A growing body of research indicates a correlation between heart disease and a lack of micronutrients. Repairable heart disease has been linked to thiamine and selenium deficiency. Micronutrients and heart disease may, nevertheless, have a more moderate association, according to recent research. This article looks at studies that looked at micronutrient consumption, supplement effectiveness, and micronutrient ingestion in heart disease patients, with an emphasis on retinol, ascorbic acid, a fat-soluble vitamin, vitamin B1, other B vitamins, cholecalciferol, folate, iron, and copper. Because aging is the leading cause of coronary heart disease, treatments intended to reduce down the aging process or improving life expectancy are distinctly different from their standards for the treatment of coronary heart disease. Altering risky life decisions which might relate to aging and coronary heart diseases, such as nicotine usage, obesity, and unique lifestyles, is increasingly become part of the quality of practice.

Effect of Amiodarone on Rabbits' Optic Nerve and the Ameliorative Effect of Vitamin E: Ftir Study

“…Vitamins intake is seen in research to be useful for individuals along with cardiovascular disease [38]. Vitamin E supplementation partially improved the optic nerve spectral analysis changes due to amiodarone injection in rabbit's since vitamin E could decrease lysosomal phospholipidosis and amiodarone toxicity [7,39].…”

Bioanalytical Rp-HPLC Method Development and Validation of Clopidogrel Bisulfate in Wistar Rat Plasma and Its Application to Pharmacokinetic Study

“…Indeed, following blood vessel injury and atherosclerotic plaque erosion, platelets adhere by their surface receptors to the subendothelial matrix, which triggers their activation and subsequent aggregation [8,9]. Activated platelets release prothrombotic mediators retained within their granules like adenosine diphosphate (ADP), serotonin, P-selectin, fibrinogen, Ca2+, and thromboxane A2 (TXA2), which further amplify platelet activation and thrombus formation [9,10]. The presence of a thrombus in an artery providing blood to a vital organ such as the heart or brain is the most common cause of acute coronary disorders, including myocardial infarction and angina [11].…”