Role of Local Trauma in Production of Cutaneous Calcinosis by Dihydrotachysterol.

Selye, H; Jean, P; Veilleux, R

doi:10.3181/00379727-104-25855

Cited by 16 publications

(5 citation statements)

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“…Experimental studies carried out by Selye et al (Grasso and Selye, 1962;Selye, 1962;Selye, Jean, and Veilleux, 1960) show that the deposition of calcium in the tissues can be the result of an interaction of local and general factors. The administration of parathyroid hormone and vitamin D derivatives results in the mobilization of the calcium from the skeleton, and in large doses these substances cause tissue calcifications without additional localizing factors.…”

Section: Discussionmentioning

confidence: 99%

“…Similar changes in the coronaries are seen in the idiopathic arterial calcification of infancy (Moran and Becker, 1959). In a calciphylactic state a mild mechanical trauma also acts as a challenger (Selye et al, 1960). Thus, a higher haemodynamic strain could probably play a similar role and promote the calcific incrustation in the arterial segments which are subject to a higher wall tension.…”

Section: Discussionmentioning

confidence: 99%

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Calcifications of the Carotid Siphon--A Common Finding in Infancy and Childhood

Meyer¹,

Lind²

1972

Archives of Disease in Childhood

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

Calcifications of the Carotid Siphon--A Common Finding in Infancy and Childhood

Meyer¹,

Lind²

1972

Archives of Disease in Childhood

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“…Experiments in dihydrotachysterol (DHT)-sensitized rats indicated that functional stress and topical trauma can produce local calcium deposits in various parts of the body [91,93,94]. In rats given DHT, enlargement of buccal and lingual bone occurred most notably at muscle insertions [95].…”

Section: Role Of Local Traumamentioning

confidence: 99%

Update on the Etiology of Tooth Resorption in Domestic Cats

Reiter

Lewis

Okuda

2005

Veterinary Clinics of North America: Small Animal Practice

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Feline odontoclastic resorptive lesions (FORL) were first recognized and histologically differentiated from caries in the 1920s [1,2]. Some anecdotal reports describing caries-like lesions at the cervical region of feline teeth followed in the 1950s and 1960s, until two microscopic studies in the 1970s again revealed that FORL were not caries but a type of tooth resorption [3,4]. A recent study showed that cats with FORL have a significantly lower urine specific gravity and significantly higher serum concentration of 25-hydroxyvitamin D (25OHD) compared with cats without FORL [5], indicating that multiple tooth resorption in domestic cats could be the manifestation of some systemic insult rather than a local cause. In this article, the histologic and radiographic appearance of FORL and certain other peculiarities of feline teeth are reviewed. An attempt is then made to compare these findings with changes of the periodontium induced by administration of excess vitamin D or vitamin D metabolites in experimental animals. Histologic and radiographic features of feline odontoclastic resorptive lesionsTooth resorption is caused by odontoclasts. Their precursors derive from hematopoietic stem cells of bone marrow or spleen and migrate from blood vessels of the alveolar bone or periodontal ligament toward the external root

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“…Moreover, among the me chanical traumas, epilation is one of the most effective means of producing cutaneous calcinosis, but the lesion is best produced when epilation is performed at the "critical period," i.e., during the last 2 days of DHT-treatment. 6…”

Section: Discussionmentioning

confidence: 99%