Risk Factors Associated with Subdural Hygroma after Decompressive Craniectomy in Patients with Traumatic Brain Injury : A Comparative Study

Abstract

GOS showed statistically significant difference in the clinical risk factors for SDG between the group with SDG and the group without SDG. Analysis of radiological factors indicated that a midline shifting exceeding 5 mm, SAH, delayed hydrocephalus, compression of basal cisterns, and tearing of the arachnoid membrane were significantly more common in patients with SDG.

Subdural hygroma following decompressive craniectomy or non-decompressive craniectomy in patients with traumatic brain injury: Clinical features and risk factors

“…The main reason is likely use of different inclusion and diagnostic criteria in the previous study; however, uniform inclusion and diagnostic criteria were used in this work. Jeon et al [26] reported that radiological factors indicate that a midline shift 45 mm, SAH, delayed hydrocephalus, compression of basal cisterns and tearing of the arachnoid membrane may be risk factors associated with SDG after DC. However, in this study, the risk factors for SDG in patients without DC, ipsilateral SDG after unilateral DC, contralateral SDG after unilateral DC and SDG after bilateral DC varied.…”

Posttraumatic Hydrocephalus after Decompressive Craniectomy in 126 Patients with Severe Traumatic Brain Injury

“…The edge of the craniectomy being closer than 25 mm to the midline; a large craniectomy; the existence, thickness, and distribution of subarachnoid hemorrhage (SAH); the existence of a subdural hygroma; the degree of hypoperfusion in the temporal lobe; and repeated operations and duration of coma may be associated with PTH. 6,10,20,[25][26][27][33][34][35][36] SAH localization, evacuation of a contusion, CSF infection, high Fisher grade, and sex are the factors Abbreviations: PTH, posttraumatic hydrocephalus. a Correlation is significant at the 0.05 level (two tailed).…”

Decompressive Craniectomy in Neurocritical Care

“…115 Disruption of the arachnoid membranes on postoperative CT strongly predicts the development of post-DC hygroma formation, suggesting that hygroma formation involves abnormal communications between the subdural and subarachnoid spaces. 116 Given its self-limited nature, most hygromas can be managed with close observation. However, patients with hygromas should be monitored closely for the development of hydrocephalus.…”