1958
DOI: 10.1016/s0022-3476(58)80039-6
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Rheumatic pneumonia

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1960
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Cited by 9 publications
(2 citation statements)
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“…Furthermore, some infections may initiate an uncontrolled inflammatory organising pneumonia process that persists after the aetiological agent has disappeared. An infectious agent may also induce a secondary noxious immunopathological process; a convincing example is rheumatic pneumonia [224,225] where, in addition to the well known cardiac complications, typical organising pneumonia has been described [225][226][227], especially by MASSON et al [227], who called the intra-alveolar buds ''bourgeons conjonctifs'' (connective tissue buds), terminology still used by some pathologists [228]. The clinical descriptions of rheumatic organising pneumonia have mentioned the ''fleeting nature'' of lung pneumonic opacities, improvement with adrenocorticotropin (ACTH), and even cited a case of ''rebound phenomenon after small doses of ACTH were discontinued'' [224].…”
Section: Determined Causes Of Organising Pneumoniamentioning
confidence: 99%
“…Furthermore, some infections may initiate an uncontrolled inflammatory organising pneumonia process that persists after the aetiological agent has disappeared. An infectious agent may also induce a secondary noxious immunopathological process; a convincing example is rheumatic pneumonia [224,225] where, in addition to the well known cardiac complications, typical organising pneumonia has been described [225][226][227], especially by MASSON et al [227], who called the intra-alveolar buds ''bourgeons conjonctifs'' (connective tissue buds), terminology still used by some pathologists [228]. The clinical descriptions of rheumatic organising pneumonia have mentioned the ''fleeting nature'' of lung pneumonic opacities, improvement with adrenocorticotropin (ACTH), and even cited a case of ''rebound phenomenon after small doses of ACTH were discontinued'' [224].…”
Section: Determined Causes Of Organising Pneumoniamentioning
confidence: 99%
“…Pulmonary disease may occur in both the first and recurrent attacks of ARF, either as part of the rheumatic process, or secondary to the cardiac lesion, congestive heart failure, uraemia or intercurrent infection 2. The incidence of pulmonary disease in ARF has greatly varied (2–60%) depending on the rigidity of the criteria used and the nature of the series 1…”
mentioning
confidence: 99%